Linked Life Data

17152715

Source:http://linkedlifedata.com/resource/pubmed/id/17152715

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:dateCreated
2006-12-8
pubmed:abstractText
Numerous empiric data prove incontestably that species-specific lifespan is under control of genetic program. At the same time the modern evolution theories of aging deny an opportunity of existence of specific genes predetermining longevity. Basic premise for non-acceptance of the aging gene concept is convincing: animals in habitat do not live long enough for aging to exert any effect on their survival. Therefore, the natural selection can not differentiate them by the longevity attribute. To solve the collision the population approach was used in this work. As known from ecological laws the innate population growth rate (r(in)), lifetime of one generation (T), and inborn fertility (R(0)) are interconnected by the next dependence: r(in) = In R(0)/T. The examining of mathematical model of population showed that r(in) value is stabilized by inter-population natural selection on the level which corresponds to environment press in the species ecological niche. Therefore, species-specific longevity and fecundity are under control of this natural selection.
pubmed:language
rus
pubmed:journal
pubmed:citationSubset
IM
pubmed:status
MEDLINE
pubmed:issn
1561-9125
pubmed:author
pubmed:issnType
Print
pubmed:volume
19
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
13-24
pubmed:dateRevised
2010-11-18
pubmed:meshHeading
pubmed:year
2006
pubmed:articleTitle
[Evolution mechanism of species-specific lifespan].
pubmed:publicationType
Journal Article, English Abstract