17151932

Source:http://linkedlifedata.com/resource/pubmed/id/17151932

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0017638, umls-concept:C0018591, umls-concept:C0025286, umls-concept:C0035647, umls-concept:C0042333, umls-concept:C0079419, umls-concept:C0678214, umls-concept:C0919524
pubmed:issue	3
pubmed:dateCreated	2007-4-17
pubmed:abstractText	P53 and ATM are central checkpoint genes involved in the repair of DNA damage after ionising irradiation, which has been associated with risk of brain tumours. Therefore, we tested the hypothesis that polymorphisms and haplotypes in p53 and ATM could be associated with glioma and meningioma risk.
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/8309335
pubmed:citationSubset	IM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/Cell Cycle Proteins, http://linkedlifedata.com/resource/pubmed/chemical/DNA-Binding Proteins, http://linkedlifedata.com/resource/pubmed/chemical/Protein-Serine-Threonine Kinases, http://linkedlifedata.com/resource/pubmed/chemical/Tumor Suppressor Protein p53, http://linkedlifedata.com/resource/pubmed/chemical/Tumor Suppressor Proteins, http://linkedlifedata.com/resource/pubmed/chemical/ataxia telangiectasia mutated...
pubmed:status	MEDLINE
pubmed:month	May
pubmed:issn	0167-594X
pubmed:author	pubmed-author:AhlbomAndersA, pubmed-author:AuvinenAnssiA, pubmed-author:Collatz-ChristensenHelleH, pubmed-author:FeychtingMariaM, pubmed-author:GrönbergHenrikH, pubmed-author:HenrikssonRogerR, pubmed-author:JohansenChristofferC, pubmed-author:KiuruAnneA, pubmed-author:LönnStefanS, pubmed-author:LindströmSaraS, pubmed-author:MalmerBeatrice SusanneBS, pubmed-author:MudieNadejdaN, pubmed-author:SalminenTiinaT, pubmed-author:SchoemakerMinouk JMJ, pubmed-author:SchwartzbaumJudyJ, pubmed-author:SwerdlowAnthony JAJ
pubmed:issnType	Print
pubmed:volume	82
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	229-37
pubmed:dateRevised	2011-11-2
pubmed:meshHeading	pubmed-meshheading:17151932-Adult, pubmed-meshheading:17151932-Aged, pubmed-meshheading:17151932-Brain Neoplasms, pubmed-meshheading:17151932-Case-Control Studies, pubmed-meshheading:17151932-Cell Cycle Proteins, pubmed-meshheading:17151932-DNA-Binding Proteins, pubmed-meshheading:17151932-Female, pubmed-meshheading:17151932-Genetic Predisposition to Disease, pubmed-meshheading:17151932-Glioma, pubmed-meshheading:17151932-Haplotypes, pubmed-meshheading:17151932-Humans, pubmed-meshheading:17151932-Male, pubmed-meshheading:17151932-Meningeal Neoplasms, pubmed-meshheading:17151932-Meningioma, pubmed-meshheading:17151932-Middle Aged, pubmed-meshheading:17151932-Polymorphism, Single Nucleotide, pubmed-meshheading:17151932-Protein-Serine-Threonine Kinases, pubmed-meshheading:17151932-Risk Factors, pubmed-meshheading:17151932-Tumor Suppressor Protein p53, pubmed-meshheading:17151932-Tumor Suppressor Proteins
pubmed:year	2007
pubmed:articleTitle	Genetic variation in p53 and ATM haplotypes and risk of glioma and meningioma.
pubmed:affiliation	Department of Radiation Sciences, Oncology, Umeå University Hospital, 901 85, Umeå, Sweden. Beatrice.malmer@onkologi.umu.se
pubmed:publicationType	Journal Article, Research Support, Non-U.S. Gov't