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rdf:type |
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lifeskim:mentions |
|
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pubmed:issue |
1
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pubmed:dateCreated |
2007-1-15
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pubmed:abstractText |
Extracellular ATP acting on the P2Y11 receptor regulates inflammatory cells. We hypothesized that polymorphisms in the receptor could influence the risk of acute myocardial infarction (AMI).
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Jan
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pubmed:issn |
0195-668X
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pubmed:author |
|
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pubmed:issnType |
Print
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pubmed:volume |
28
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
13-8
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pubmed:dateRevised |
2010-1-14
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pubmed:meshHeading |
pubmed-meshheading:17135283-Adult,
pubmed-meshheading:17135283-C-Reactive Protein,
pubmed-meshheading:17135283-Case-Control Studies,
pubmed-meshheading:17135283-Female,
pubmed-meshheading:17135283-Genotype,
pubmed-meshheading:17135283-Homozygote,
pubmed-meshheading:17135283-Humans,
pubmed-meshheading:17135283-Male,
pubmed-meshheading:17135283-Myocardial Infarction,
pubmed-meshheading:17135283-Odds Ratio,
pubmed-meshheading:17135283-Polymorphism, Genetic,
pubmed-meshheading:17135283-Receptors, Purinergic P2
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pubmed:year |
2007
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pubmed:articleTitle |
Increased risk of acute myocardial infarction and elevated levels of C-reactive protein in carriers of the Thr-87 variant of the ATP receptor P2Y11.
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pubmed:affiliation |
Department of Cardiology, Lund University Hospital, SE-221 85 Lund, Sweden.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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