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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
2
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pubmed:dateCreated |
2007-2-12
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pubmed:abstractText |
Although the mechanism that underlies aspirin hypersensitivity is not completely understood, an IgE-mediated response was reported for a patient with aspirin-intolerant chronic urticaria (AICU).
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pubmed:commentsCorrections |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Feb
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pubmed:issn |
0091-6749
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
119
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
449-56
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pubmed:dateRevised |
2008-11-21
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pubmed:meshHeading |
pubmed-meshheading:17125826-Aspirin,
pubmed-meshheading:17125826-Basophils,
pubmed-meshheading:17125826-Chronic Disease,
pubmed-meshheading:17125826-DNA-Binding Proteins,
pubmed-meshheading:17125826-Drug Hypersensitivity,
pubmed-meshheading:17125826-Histamine Release,
pubmed-meshheading:17125826-Humans,
pubmed-meshheading:17125826-Polymorphism, Single Nucleotide,
pubmed-meshheading:17125826-Promoter Regions, Genetic,
pubmed-meshheading:17125826-Receptors, IgE,
pubmed-meshheading:17125826-Transcription Factors,
pubmed-meshheading:17125826-Urticaria
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pubmed:year |
2007
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pubmed:articleTitle |
Significant association of FcepsilonRIalpha promoter polymorphisms with aspirin-intolerant chronic urticaria.
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pubmed:affiliation |
Department of Allergy and Rheumatology, Ajou University School of Medicine, Suwon, Korea.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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