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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
12
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pubmed:dateCreated |
2006-11-14
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pubmed:abstractText |
Studies of the pathological mechanisms of food allergy have been impeded by the lack of relevant animal models. The purpose of this study was to develop a physiological model of food allergy that was not dependent on immunostimulatory adjuvants.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Dec
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pubmed:issn |
0036-5521
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pubmed:author |
|
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pubmed:issnType |
Print
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pubmed:volume |
41
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1405-13
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pubmed:meshHeading |
pubmed-meshheading:17101571-Animals,
pubmed-meshheading:17101571-Chymases,
pubmed-meshheading:17101571-Diarrhea,
pubmed-meshheading:17101571-Disease Models, Animal,
pubmed-meshheading:17101571-Female,
pubmed-meshheading:17101571-Food Hypersensitivity,
pubmed-meshheading:17101571-Immunization,
pubmed-meshheading:17101571-Immunoglobulin E,
pubmed-meshheading:17101571-Immunoglobulin G,
pubmed-meshheading:17101571-Immunohistochemistry,
pubmed-meshheading:17101571-Mice,
pubmed-meshheading:17101571-Mice, Inbred BALB C,
pubmed-meshheading:17101571-Ovalbumin
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pubmed:year |
2006
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pubmed:articleTitle |
Murine model of food allergy after epicutaneous sensitization: role of mucosal mast cell protease-1.
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pubmed:affiliation |
Department of Viral Diseases and Immunology, Laboratory of Immunology, National Public Health Institute, Helsinki, Finland. kirsi.vaali@ktl.fi
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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