Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
1
pubmed:dateCreated
2006-12-29
pubmed:abstractText
Recent studies have identified the involvement of inhibitor IkappaB kinase (IKK) in the pathogenesis of insulin resistance. To investigate the mechanism involved, we examined the role of nuclear factor kappaB (NF-kappaB), the distal target of IKK, in hepatic glucose metabolism.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
http://linkedlifedata.com/resource/pubmed/chemical/AMP-Activated Protein Kinases, http://linkedlifedata.com/resource/pubmed/chemical/Creb1 protein, mouse, http://linkedlifedata.com/resource/pubmed/chemical/Cyclic AMP Response..., http://linkedlifedata.com/resource/pubmed/chemical/Glucose, http://linkedlifedata.com/resource/pubmed/chemical/Glycogen, http://linkedlifedata.com/resource/pubmed/chemical/I kappa B beta protein, http://linkedlifedata.com/resource/pubmed/chemical/I-kappa B Proteins, http://linkedlifedata.com/resource/pubmed/chemical/Multienzyme Complexes, http://linkedlifedata.com/resource/pubmed/chemical/NF-kappa B, http://linkedlifedata.com/resource/pubmed/chemical/Ppargc1a protein, mouse, http://linkedlifedata.com/resource/pubmed/chemical/Protein-Serine-Threonine Kinases, http://linkedlifedata.com/resource/pubmed/chemical/STAT3 Transcription Factor, http://linkedlifedata.com/resource/pubmed/chemical/Stat3 protein, mouse, http://linkedlifedata.com/resource/pubmed/chemical/Trans-Activators, http://linkedlifedata.com/resource/pubmed/chemical/Triglycerides
pubmed:status
MEDLINE
pubmed:month
Jan
pubmed:issn
0012-186X
pubmed:author
pubmed:issnType
Print
pubmed:volume
50
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
131-41
pubmed:dateRevised
2008-11-21
pubmed:meshHeading
pubmed-meshheading:17093946-AMP-Activated Protein Kinases, pubmed-meshheading:17093946-Adenoviridae, pubmed-meshheading:17093946-Animals, pubmed-meshheading:17093946-Cyclic AMP Response Element-Binding Protein, pubmed-meshheading:17093946-Diabetes Mellitus, pubmed-meshheading:17093946-Disease Models, Animal, pubmed-meshheading:17093946-Female, pubmed-meshheading:17093946-Glucose, pubmed-meshheading:17093946-Glycogen, pubmed-meshheading:17093946-I-kappa B Proteins, pubmed-meshheading:17093946-Insulin Resistance, pubmed-meshheading:17093946-Liver, pubmed-meshheading:17093946-Mice, pubmed-meshheading:17093946-Mice, Inbred C57BL, pubmed-meshheading:17093946-Multienzyme Complexes, pubmed-meshheading:17093946-NF-kappa B, pubmed-meshheading:17093946-Protein-Serine-Threonine Kinases, pubmed-meshheading:17093946-STAT3 Transcription Factor, pubmed-meshheading:17093946-Trans-Activators, pubmed-meshheading:17093946-Triglycerides
pubmed:year
2007
pubmed:articleTitle
Amelioration of glucose tolerance by hepatic inhibition of nuclear factor kappaB in db/db mice.
pubmed:affiliation
Department of Medicine, Metabolism and Endocrinology, Juntendo University School of Medicine, 2-1-1 Hongo, Bunkyo-ku, Tokyo, 113-8421, Japan.
pubmed:publicationType
Journal Article