Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
5
pubmed:dateCreated
2006-11-20
pubmed:abstractText
The aim of this case-control study was to test the hypothesis that polymorphisms of the microsomal epoxide hydroxylase (mEH) and glutathione S-transferase (GST) genes modulate the susceptibility to esophageal adenocarcinoma (EADC) associated with smoking.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:issn
0361-090X
pubmed:author
pubmed:issnType
Print
pubmed:volume
30
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
423-31
pubmed:dateRevised
2009-11-19
pubmed:meshHeading
pubmed-meshheading:17064856-Adenocarcinoma, pubmed-meshheading:17064856-Alleles, pubmed-meshheading:17064856-Barrett Esophagus, pubmed-meshheading:17064856-Case-Control Studies, pubmed-meshheading:17064856-Epoxide Hydrolases, pubmed-meshheading:17064856-Esophageal Neoplasms, pubmed-meshheading:17064856-Female, pubmed-meshheading:17064856-Gastroesophageal Reflux, pubmed-meshheading:17064856-Genetic Predisposition to Disease, pubmed-meshheading:17064856-Genotype, pubmed-meshheading:17064856-Glutathione S-Transferase pi, pubmed-meshheading:17064856-Glutathione Transferase, pubmed-meshheading:17064856-Humans, pubmed-meshheading:17064856-Male, pubmed-meshheading:17064856-Middle Aged, pubmed-meshheading:17064856-Polymorphism, Genetic, pubmed-meshheading:17064856-Risk Factors, pubmed-meshheading:17064856-Smoking
pubmed:year
2006
pubmed:articleTitle
Genetic polymorphisms of microsomal epoxide hydroxylase and glutathione S-transferases M1, T1 and P1, interactions with smoking, and risk for esophageal (Barrett) adenocarcinoma.
pubmed:affiliation
Department of Surgery, University of Saskatchewan, Royal University Hospital, Saskatoon, Sask, Canada. alan.casson@usask.ca
pubmed:publicationType
Journal Article, Comparative Study, Research Support, Non-U.S. Gov't