Source:http://linkedlifedata.com/resource/pubmed/id/17001703
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
9
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pubmed:dateCreated |
2006-10-2
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pubmed:abstractText |
Amniotic bands can cause disruption of the cranial end of the developing fetus, leading in some cases to a neural tube closure defect. Although recurrence for unaffected parents of an affected child with a defect in which the neural tube closed normally but was subsequently disrupted by amniotic bands is negligible; for a primary defect in closure of the neural tube to which amnion has subsequently adhered, recurrence risk is 1.7%. In that primary defects of neural tube closure are characterized by typical abnormalities of the base of the skull, evaluation of the cranial base in such fetuses provides an approach for making a distinction between these 2 mechanisms. This distinction has implications regarding recurrence risk.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:status |
MEDLINE
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pubmed:month |
Sep
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pubmed:issn |
1542-0752
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
76
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
688-91
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pubmed:meshHeading | |
pubmed:year |
2006
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pubmed:articleTitle |
Evaluation of the cranial base in amnion rupture sequence involving the anterior neural tube: implications regarding recurrence risk.
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pubmed:affiliation |
Department of Pediatrics, University of California San Diego, School of Medicine, La Jolla, California, USA. klyons@ucsd.edu
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pubmed:publicationType |
Journal Article,
Comparative Study
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