| pubmed-article:16973968 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:16973968 | lifeskim:mentions | umls-concept:C1801960 | lld:lifeskim |
| pubmed-article:16973968 | lifeskim:mentions | umls-concept:C0529330 | lld:lifeskim |
| pubmed-article:16973968 | lifeskim:mentions | umls-concept:C0015376 | lld:lifeskim |
| pubmed-article:16973968 | lifeskim:mentions | umls-concept:C0205160 | lld:lifeskim |
| pubmed-article:16973968 | lifeskim:mentions | umls-concept:C0851285 | lld:lifeskim |
| pubmed-article:16973968 | lifeskim:mentions | umls-concept:C0332206 | lld:lifeskim |
| pubmed-article:16973968 | pubmed:issue | 12 | lld:pubmed |
| pubmed-article:16973968 | pubmed:dateCreated | 2006-11-19 | lld:pubmed |
| pubmed-article:16973968 | pubmed:abstractText | Permeability of blood vessels is essential for tissue homeostasis. However, disorganized hyperpermeability leads to progression of diseases. Vascular endothelial growth factor-A (VEGF) is a key regulator for leakiness of blood vessels and it has been reported that VEGF-mediated hyperpermeability was suppressed by angiopoietin-1 (Ang1). We found that Angiotensin-converting enzyme (ACE) was downregulated in endothelial cells by Ang1. ACE converts angiotensin I to angiotensin II (AII). Here, we studied the relationship between VEGF and AII relative to vascular permeability. | lld:pubmed |
| pubmed-article:16973968 | pubmed:language | eng | lld:pubmed |
| pubmed-article:16973968 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16973968 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:16973968 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16973968 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16973968 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16973968 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:16973968 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16973968 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16973968 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16973968 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:16973968 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16973968 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16973968 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16973968 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:16973968 | pubmed:issn | 1524-4636 | lld:pubmed |
| pubmed-article:16973968 | pubmed:author | pubmed-author:TakakuraNobuy... | lld:pubmed |
| pubmed-article:16973968 | pubmed:author | pubmed-author:SanoHidetoH | lld:pubmed |
| pubmed-article:16973968 | pubmed:author | pubmed-author:KidoyaHiroyas... | lld:pubmed |
| pubmed-article:16973968 | pubmed:author | pubmed-author:HosokawaKohei... | lld:pubmed |
| pubmed-article:16973968 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:16973968 | pubmed:volume | 26 | lld:pubmed |
| pubmed-article:16973968 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16973968 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16973968 | pubmed:pagination | 2673-80 | lld:pubmed |
| pubmed-article:16973968 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
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| pubmed-article:16973968 | pubmed:year | 2006 | lld:pubmed |
| pubmed-article:16973968 | pubmed:articleTitle | Negative regulation of VEGF-induced vascular leakage by blockade of angiotensin II type 1 receptor. | lld:pubmed |
| pubmed-article:16973968 | pubmed:affiliation | Department of Stem Cell Biology, Cancer Research Institute, Kanazawa University, 13-1, Takara-machi, Kanazawa, Ishikawa, 920-0934 Japan. | lld:pubmed |
| pubmed-article:16973968 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16973968 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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