Source:http://linkedlifedata.com/resource/pubmed/id/16960441
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
5
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pubmed:dateCreated |
2006-12-28
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pubmed:abstractText |
The combination of diabetes and hypertension increases the incidence and severity of kidney disease in an additive manner. Inflammatory and oxidative stress mechanisms contribute to renal damage in both diabetes and hypertension. Therefore, we investigated whether renal macrophage infiltration and oxidative stress events are additive from the beginning in diabetic animals with coexisting hypertension.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:status |
MEDLINE
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pubmed:issn |
0250-8095
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pubmed:author | |
pubmed:copyrightInfo |
Copyright (c) 2006 S. Karger AG, Basel.
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pubmed:issnType |
Print
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pubmed:volume |
26
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
415-22
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pubmed:meshHeading |
pubmed-meshheading:16960441-Animals,
pubmed-meshheading:16960441-Cell Movement,
pubmed-meshheading:16960441-Diabetes Mellitus, Experimental,
pubmed-meshheading:16960441-Diabetic Nephropathies,
pubmed-meshheading:16960441-Hypertension,
pubmed-meshheading:16960441-Macrophages,
pubmed-meshheading:16960441-Male,
pubmed-meshheading:16960441-Oxidative Stress,
pubmed-meshheading:16960441-Rats,
pubmed-meshheading:16960441-Rats, Inbred SHR,
pubmed-meshheading:16960441-Rats, Inbred WKY
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pubmed:year |
2006
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pubmed:articleTitle |
Hypertension induces oxidative stress but not macrophage infiltration in the kidney in the early stage of experimental diabetes mellitus.
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pubmed:affiliation |
Renal Pathophysiology Laboratory, Division of Nephrology, Department of Internal Medicine, Faculty of Medical Sciences, State University of Campinas (UNICAMP), Campinas, São Paulo, Brazil.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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