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| pubmed-article:16956821 | lifeskim:mentions | umls-concept:C0035647 | lld:lifeskim |
| pubmed-article:16956821 | lifeskim:mentions | umls-concept:C0024305 | lld:lifeskim |
| pubmed-article:16956821 | lifeskim:mentions | umls-concept:C0242606 | lld:lifeskim |
| pubmed-article:16956821 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
| pubmed-article:16956821 | lifeskim:mentions | umls-concept:C0007367 | lld:lifeskim |
| pubmed-article:16956821 | lifeskim:mentions | umls-concept:C0017822 | lld:lifeskim |
| pubmed-article:16956821 | lifeskim:mentions | umls-concept:C0038838 | lld:lifeskim |
| pubmed-article:16956821 | lifeskim:mentions | umls-concept:C1882417 | lld:lifeskim |
| pubmed-article:16956821 | pubmed:issue | 9 | lld:pubmed |
| pubmed-article:16956821 | pubmed:dateCreated | 2006-9-7 | lld:pubmed |
| pubmed-article:16956821 | pubmed:abstractText | The risk of non-Hodgkin's lymphoma (NHL) has been associated with inflammation. One possible mechanism may involve oxidative stress as reactive oxygen species (ROS) can generate pro-inflammatory signals. Anti-oxidant enzymes including superoxide dismutase, glutathione peroxidase and catalase protect against the harmful effects of ROS. Genetic variation in the genes coding for these enzymes (SOD2, GPX1, and CAT, respectively) alters ROS production and therefore may provide a mechanism for the relationship between inflammation and NHL. | lld:pubmed |
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| pubmed-article:16956821 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:16956821 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:16956821 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16956821 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:16956821 | pubmed:issn | 1592-8721 | lld:pubmed |
| pubmed-article:16956821 | pubmed:author | pubmed-author:HollyElizabet... | lld:pubmed |
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| pubmed-article:16956821 | pubmed:author | pubmed-author:LightfootTrac... | lld:pubmed |
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| pubmed-article:16956821 | pubmed:author | pubmed-author:SmithAlex GAG | lld:pubmed |
| pubmed-article:16956821 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:16956821 | pubmed:volume | 91 | lld:pubmed |
| pubmed-article:16956821 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16956821 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16956821 | pubmed:pagination | 1222-7 | lld:pubmed |
| pubmed-article:16956821 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:16956821 | pubmed:year | 2006 | lld:pubmed |
| pubmed-article:16956821 | pubmed:articleTitle | Polymorphisms in the oxidative stress genes, superoxide dismutase, glutathione peroxidase and catalase and risk of non-Hodgkin's lymphoma. | lld:pubmed |
| pubmed-article:16956821 | pubmed:affiliation | Epidemiology & Genetics Unit, University of York, York, YO10 5DD, UK. tracy.lightfoot@egu.york.ac.uk | lld:pubmed |
| pubmed-article:16956821 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16956821 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:16956821 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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