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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
7
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pubmed:dateCreated |
2006-9-5
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pubmed:abstractText |
In congestive heart failure (CHF), an activation of the cardiac sympathetic nervous system results in depleted cardiac norepinephrine (NE) stores. The underlying regulatory mechanisms are discussed controversially and were investigated in the present study in CHF resulting from volume overload.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
|
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pubmed:status |
MEDLINE
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pubmed:month |
Sep
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pubmed:issn |
1532-8414
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pubmed:author |
|
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pubmed:issnType |
Electronic
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pubmed:volume |
12
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
577-83
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pubmed:dateRevised |
2007-11-15
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pubmed:meshHeading |
pubmed-meshheading:16952792-Animals,
pubmed-meshheading:16952792-Aorta,
pubmed-meshheading:16952792-Arteriovenous Shunt, Surgical,
pubmed-meshheading:16952792-Cardiomegaly,
pubmed-meshheading:16952792-Heart Failure,
pubmed-meshheading:16952792-Homeostasis,
pubmed-meshheading:16952792-Hypertension,
pubmed-meshheading:16952792-Male,
pubmed-meshheading:16952792-Myocardium,
pubmed-meshheading:16952792-Nerve Endings,
pubmed-meshheading:16952792-Norepinephrine,
pubmed-meshheading:16952792-Rats,
pubmed-meshheading:16952792-Rats, Wistar,
pubmed-meshheading:16952792-Sympathetic Nervous System,
pubmed-meshheading:16952792-Venae Cavae
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pubmed:year |
2006
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pubmed:articleTitle |
Preserved norepinephrine reuptake but reduced sympathetic nerve endings in hypertrophic volume-overloaded rat hearts.
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pubmed:affiliation |
Department of Cardiology, Medical University of Heidelberg, Heidelberg, Germany.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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