Linked Life Data

16952202

Source:http://linkedlifedata.com/resource/pubmed/id/16952202

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
3
pubmed:dateCreated
2007-3-5
pubmed:abstractText
Decreased beta-cell mass, mainly due to apoptosis, is crucial for the development and progression of type 2 diabetes. Chronic exposure to high glucose levels is a probable underlying mechanism, whereas the role of oral anti-diabetic agents (sulphonylureas in particular) is still unsettled.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Mar
pubmed:issn
1520-7552
pubmed:author
pubmed:copyrightInfo
(c) 2006 John Wiley & Sons, Ltd.
pubmed:issnType
Print
pubmed:volume
23
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
234-8
pubmed:dateRevised
2011-11-17
pubmed:meshHeading
pubmed:year
2007
pubmed:articleTitle
Gliclazide protects human islet beta-cells from apoptosis induced by intermittent high glucose.
pubmed:affiliation
Department of Endocrinology and Metabolism, Metabolic Unit, University of Pisa, Pisa, Italy.
pubmed:publicationType
Journal Article