| pubmed-article:16921253 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:16921253 | lifeskim:mentions | umls-concept:C1801960 | lld:lifeskim |
| pubmed-article:16921253 | lifeskim:mentions | umls-concept:C0596290 | lld:lifeskim |
| pubmed-article:16921253 | lifeskim:mentions | umls-concept:C0132555 | lld:lifeskim |
| pubmed-article:16921253 | lifeskim:mentions | umls-concept:C0597298 | lld:lifeskim |
| pubmed-article:16921253 | lifeskim:mentions | umls-concept:C1517499 | lld:lifeskim |
| pubmed-article:16921253 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:16921253 | pubmed:dateCreated | 2006-9-19 | lld:pubmed |
| pubmed-article:16921253 | pubmed:abstractText | Many vascular diseases are associated with reduced nitric oxide (NO) bioavailability. Nitric oxide synthase (NOS) gene therapy to the vasculature is a possible treatment for vascular disease as a means of increasing NO bioavailability, and this may be achieved using any of the NOS isoforms. The aim of our study was to compare the effects of adenoviral-mediated overexpression of the most commonly used NOS isoforms eNOS and iNOS on vascular cell proliferation. | lld:pubmed |
| pubmed-article:16921253 | pubmed:language | eng | lld:pubmed |
| pubmed-article:16921253 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16921253 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:16921253 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:16921253 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16921253 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16921253 | pubmed:issn | 1018-1172 | lld:pubmed |
| pubmed-article:16921253 | pubmed:author | pubmed-author:HynesSean OSO | lld:pubmed |
| pubmed-article:16921253 | pubmed:author | pubmed-author:O'BrienTimoth... | lld:pubmed |
| pubmed-article:16921253 | pubmed:author | pubmed-author:SharifFaisalF | lld:pubmed |
| pubmed-article:16921253 | pubmed:author | pubmed-author:CooneyRonanR | lld:pubmed |
| pubmed-article:16921253 | pubmed:author | pubmed-author:DuffyAoife... | lld:pubmed |
| pubmed-article:16921253 | pubmed:copyrightInfo | Copyright (c) 2006 S. Karger AG, Basel. | lld:pubmed |
| pubmed-article:16921253 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:16921253 | pubmed:volume | 43 | lld:pubmed |
| pubmed-article:16921253 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16921253 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16921253 | pubmed:pagination | 462-72 | lld:pubmed |
| pubmed-article:16921253 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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| pubmed-article:16921253 | pubmed:meshHeading | pubmed-meshheading:16921253... | lld:pubmed |
| pubmed-article:16921253 | pubmed:year | 2006 | lld:pubmed |
| pubmed-article:16921253 | pubmed:articleTitle | Adenoviral-mediated gene transfer of nitric oxide synthase isoforms and vascular cell proliferation. | lld:pubmed |
| pubmed-article:16921253 | pubmed:affiliation | Regenerative Medicine Institute (REMEDI), National Centre for Biomedical Engineering Sciences (NCBES), National University of Ireland, Galway, Ireland. timothy.obrien@nuigalway.ie | lld:pubmed |
| pubmed-article:16921253 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16921253 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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