16899302

Source:http://linkedlifedata.com/resource/pubmed/id/16899302

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0011155, umls-concept:C0017262, umls-concept:C0026769, umls-concept:C0039194, umls-concept:C0185117, umls-concept:C1332700, umls-concept:C1332714, umls-concept:C1539477, umls-concept:C2911684
pubmed:issue	1-2
pubmed:dateCreated	2006-11-10
pubmed:abstractText	To evaluate whether T cells expressing CCR5 and CXCR3 from multiple sclerosis (MS) patients are more resistant to apoptosis.
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/8109498
pubmed:citationSubset	IM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/Antigens, CD, http://linkedlifedata.com/resource/pubmed/chemical/Antigens, CD95, http://linkedlifedata.com/resource/pubmed/chemical/Antigens, Differentiation..., http://linkedlifedata.com/resource/pubmed/chemical/CD69 antigen, http://linkedlifedata.com/resource/pubmed/chemical/Fas Ligand Protein, http://linkedlifedata.com/resource/pubmed/chemical/Lectins, C-Type, http://linkedlifedata.com/resource/pubmed/chemical/Proto-Oncogene Proteins c-bcl-2, http://linkedlifedata.com/resource/pubmed/chemical/Receptors, CCR5, http://linkedlifedata.com/resource/pubmed/chemical/TNF Receptor-Associated Death..., http://linkedlifedata.com/resource/pubmed/chemical/Tradd protein, mouse, http://linkedlifedata.com/resource/pubmed/chemical/bcl-2-Associated X Protein
pubmed:status	MEDLINE
pubmed:month	Nov
pubmed:issn	0165-5728
pubmed:author	pubmed-author:Al-ZayatHammadH, pubmed-author:ComabellaManuelM, pubmed-author:ErbJulieJ, pubmed-author:GoertschesRobertR, pubmed-author:Issazadeh-NavikasShohrehS, pubmed-author:MartinRolandR, pubmed-author:MontalbanXavierX
pubmed:issnType	Print
pubmed:volume	180
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	147-58
pubmed:dateRevised	2010-11-18
pubmed:meshHeading	pubmed-meshheading:16899302-Adult, pubmed-meshheading:16899302-Aged, pubmed-meshheading:16899302-Antigens, CD, pubmed-meshheading:16899302-Antigens, CD95, pubmed-meshheading:16899302-Antigens, Differentiation, T-Lymphocyte, pubmed-meshheading:16899302-Apoptosis, pubmed-meshheading:16899302-Autoimmunity, pubmed-meshheading:16899302-CD4-Positive T-Lymphocytes, pubmed-meshheading:16899302-Cell Survival, pubmed-meshheading:16899302-Chemotaxis, Leukocyte, pubmed-meshheading:16899302-Down-Regulation, pubmed-meshheading:16899302-Fas Ligand Protein, pubmed-meshheading:16899302-Female, pubmed-meshheading:16899302-Humans, pubmed-meshheading:16899302-Immunity, Cellular, pubmed-meshheading:16899302-Lectins, C-Type, pubmed-meshheading:16899302-Male, pubmed-meshheading:16899302-Middle Aged, pubmed-meshheading:16899302-Multiple Sclerosis, pubmed-meshheading:16899302-Proto-Oncogene Proteins c-bcl-2, pubmed-meshheading:16899302-Receptors, CCR5, pubmed-meshheading:16899302-TNF Receptor-Associated Death Domain Protein, pubmed-meshheading:16899302-bcl-2-Associated X Protein
pubmed:year	2006
pubmed:articleTitle	Deficient Fas expression by CD4+ CCR5+ T cells in multiple sclerosis.
pubmed:affiliation	Unitat de Neuroimmunologia Clínica, Hospital Universitari Vall d'Hebron, Universitat Autònoma de Barcelona, Barcelona, Spain.
pubmed:publicationType	Journal Article, Research Support, Non-U.S. Gov't