| pubmed-article:16866884 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:16866884 | lifeskim:mentions | umls-concept:C0332307 | lld:lifeskim |
| pubmed-article:16866884 | lifeskim:mentions | umls-concept:C0011854 | lld:lifeskim |
| pubmed-article:16866884 | lifeskim:mentions | umls-concept:C0021920 | lld:lifeskim |
| pubmed-article:16866884 | lifeskim:mentions | umls-concept:C0332281 | lld:lifeskim |
| pubmed-article:16866884 | lifeskim:mentions | umls-concept:C1413226 | lld:lifeskim |
| pubmed-article:16866884 | lifeskim:mentions | umls-concept:C1882417 | lld:lifeskim |
| pubmed-article:16866884 | lifeskim:mentions | umls-concept:C1707076 | lld:lifeskim |
| pubmed-article:16866884 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:16866884 | pubmed:dateCreated | 2006-7-26 | lld:pubmed |
| pubmed-article:16866884 | pubmed:abstractText | Type 1 diabetes is an autoimmune heterogeneous disease that is determined by environmental and genetic factors. A possible retroviral etiology has been inferred from the observation that human endogenous retrovirus (HERV)-K18 encoding a superantigen (SAg) has a polymorphism associated with this disease. Type 1 diabetes families from Germany and Belgium were genotyped for the novel HERV-8914 (303 families) and for the known HERV-8594 (284 families) polymorphisms within the SAg-coding region on the HERV-K18. Case-control analysis was performed for the HERV-8914 polymorphism (506 patients) and for the HERV-8594 polymorphism (370 patients) and compared with 350 German controls. Haplotypes were constructed. Additionally, a microsatellite within the CD48 gene was analyzed in German type 1 diabetes families (n=125) as well as in patients (n=375) and in healthy controls (n=350). No association was found for HERV-K18 polymorphisms or the CA repeat within the CD48 gene with type 1 diabetes mellitus either in families or by comparing patients and controls. In conclusion, we cannot confirm a role of HERV-K18 polymorphisms -HERV-8914 and HERV-8594- or of the CD48 CA repeat for type 1 diabetes susceptibility. | lld:pubmed |
| pubmed-article:16866884 | pubmed:language | eng | lld:pubmed |
| pubmed-article:16866884 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16866884 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:16866884 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16866884 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16866884 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:16866884 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16866884 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16866884 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:16866884 | pubmed:issn | 0001-2815 | lld:pubmed |
| pubmed-article:16866884 | pubmed:author | pubmed-author:BadenhoopKK | lld:pubmed |
| pubmed-article:16866884 | pubmed:author | pubmed-author:SeifriedEE | lld:pubmed |
| pubmed-article:16866884 | pubmed:author | pubmed-author:Van der... | lld:pubmed |
| pubmed-article:16866884 | pubmed:author | pubmed-author:SeidaHH | lld:pubmed |
| pubmed-article:16866884 | pubmed:author | pubmed-author:HerwigJJ | lld:pubmed |
| pubmed-article:16866884 | pubmed:author | pubmed-author:Van AutreveJJ | lld:pubmed |
| pubmed-article:16866884 | pubmed:author | pubmed-author:Ramos-LopezEE | lld:pubmed |
| pubmed-article:16866884 | pubmed:author | pubmed-author:GhebruSS | lld:pubmed |
| pubmed-article:16866884 | pubmed:author | pubmed-author:AminkengFF | lld:pubmed |
| pubmed-article:16866884 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:16866884 | pubmed:volume | 68 | lld:pubmed |
| pubmed-article:16866884 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16866884 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16866884 | pubmed:pagination | 147-52 | lld:pubmed |
| pubmed-article:16866884 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:16866884 | pubmed:meshHeading | pubmed-meshheading:16866884... | lld:pubmed |
| pubmed-article:16866884 | pubmed:meshHeading | pubmed-meshheading:16866884... | lld:pubmed |
| pubmed-article:16866884 | pubmed:year | 2006 | lld:pubmed |
| pubmed-article:16866884 | pubmed:articleTitle | Neither an intronic CA repeat within the CD48 gene nor the HERV-K18 polymorphisms are associated with type 1 diabetes. | lld:pubmed |
| pubmed-article:16866884 | pubmed:affiliation | Department of Internal Medicine I, Division of Endocrinology, Diabetes and Metabolism, University Hospital Frankfurt, Frankfurt am Main, Germany. | lld:pubmed |
| pubmed-article:16866884 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16866884 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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