Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
7
pubmed:dateCreated
2006-6-26
pubmed:abstractText
To identify new mouse models for studying roles of alphaAlpha-crystallin in vivo and to investigate why and how different mutations of the alphaAlpha-crystallin gene lead to dominant or recessive cataracts.
pubmed:grant
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Jul
pubmed:issn
0146-0404
pubmed:author
pubmed:issnType
Print
pubmed:volume
47
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
3004-10
pubmed:dateRevised
2010-11-18
pubmed:meshHeading
pubmed-meshheading:16799046-Actins, pubmed-meshheading:16799046-Animals, pubmed-meshheading:16799046-Arginine, pubmed-meshheading:16799046-Cataract, pubmed-meshheading:16799046-Chromosome Mapping, pubmed-meshheading:16799046-Disease Models, Animal, pubmed-meshheading:16799046-Electrophoresis, Gel, Two-Dimensional, pubmed-meshheading:16799046-Epithelial Cells, pubmed-meshheading:16799046-Female, pubmed-meshheading:16799046-Genes, Dominant, pubmed-meshheading:16799046-Genes, Recessive, pubmed-meshheading:16799046-Genetic Linkage, pubmed-meshheading:16799046-Green Fluorescent Proteins, pubmed-meshheading:16799046-Lens, Crystalline, pubmed-meshheading:16799046-Male, pubmed-meshheading:16799046-Mice, pubmed-meshheading:16799046-Mice, Inbred C3H, pubmed-meshheading:16799046-Mice, Inbred C57BL, pubmed-meshheading:16799046-Mitochondria, pubmed-meshheading:16799046-Phenotype, pubmed-meshheading:16799046-Point Mutation, pubmed-meshheading:16799046-Tyrosine, pubmed-meshheading:16799046-alpha-Crystallin A Chain
pubmed:year
2006
pubmed:articleTitle
Arginine 54 and Tyrosine 118 residues of {alpha}A-crystallin are crucial for lens formation and transparency.
pubmed:affiliation
School of Optometry and Vision Science Program, University of California, Berkeley, Berkeley, California 94720-2020, USA.
pubmed:publicationType
Journal Article, Research Support, N.I.H., Extramural