Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
5
pubmed:dateCreated
2006-10-18
pubmed:abstractText
In chronic kidney disease (CKD) patients, serum concentration of type I collagen N-telopeptide (S-NTX) increases as renal function declines because of perturbed bone metabolism by renal dysfunction itself and impaired urinary excretion of NTX. Glucocorticoid (GC), which is often used for the treatment of kidney disease, may enhance bone resorption. We studied the bone resorption state in GC-treated CKD patients and the effects of bisphosphonate on S-NTX.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Nov
pubmed:issn
8756-3282
pubmed:author
pubmed:issnType
Print
pubmed:volume
39
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
1067-72
pubmed:meshHeading
pubmed-meshheading:16781904-Adolescent, pubmed-meshheading:16781904-Adult, pubmed-meshheading:16781904-Aged, pubmed-meshheading:16781904-Aged, 80 and over, pubmed-meshheading:16781904-Biological Markers, pubmed-meshheading:16781904-Body Mass Index, pubmed-meshheading:16781904-Bone Density, pubmed-meshheading:16781904-Bone Density Conservation Agents, pubmed-meshheading:16781904-Bone Resorption, pubmed-meshheading:16781904-Chronic Disease, pubmed-meshheading:16781904-Collagen Type I, pubmed-meshheading:16781904-Creatinine, pubmed-meshheading:16781904-Cross-Sectional Studies, pubmed-meshheading:16781904-Diphosphonates, pubmed-meshheading:16781904-Etidronic Acid, pubmed-meshheading:16781904-Female, pubmed-meshheading:16781904-Glucocorticoids, pubmed-meshheading:16781904-Humans, pubmed-meshheading:16781904-Kidney Diseases, pubmed-meshheading:16781904-Male, pubmed-meshheading:16781904-Middle Aged, pubmed-meshheading:16781904-Peptides
pubmed:year
2006
pubmed:articleTitle
Serum NTX is a practical marker for assessing antiresorptive therapy for glucocorticoid treated patients with chronic kidney disease.
pubmed:affiliation
Department of Internal Medicine, Osaka University School of Medicine, Box A8, 2-2 Yamada-oka, Suita 565-0871, Japan.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't