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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
5
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pubmed:dateCreated |
2006-5-12
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pubmed:abstractText |
Oxidation of low density lipoproteins is an initial step of atherogenesis that generates pro-inflammatory phospholipids, including platelet-activating factor (PAF). PAF is degraded by PAF-acetylhydrolase (PAF-AH), which has been postulated to be a risk factor for myocardial infarction (MI). The role of PAF-AH for the onset of premature MI is unclear.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
May
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pubmed:issn |
1538-7933
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
4
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1023-8
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:16689754-1-Alkyl-2-acetylglycerophosphocholine Esterase,
pubmed-meshheading:16689754-Adult,
pubmed-meshheading:16689754-Age of Onset,
pubmed-meshheading:16689754-Cohort Studies,
pubmed-meshheading:16689754-Exons,
pubmed-meshheading:16689754-Female,
pubmed-meshheading:16689754-Genotype,
pubmed-meshheading:16689754-Humans,
pubmed-meshheading:16689754-Logistic Models,
pubmed-meshheading:16689754-Male,
pubmed-meshheading:16689754-Middle Aged,
pubmed-meshheading:16689754-Myocardial Infarction,
pubmed-meshheading:16689754-Phospholipases A,
pubmed-meshheading:16689754-Polymorphism, Genetic,
pubmed-meshheading:16689754-Risk Factors
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pubmed:year |
2006
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pubmed:articleTitle |
Platelet-activating factor-acetylhydrolase A379V (exon 11) gene polymorphism is an independent and functional risk factor for premature myocardial infarction.
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pubmed:affiliation |
Division of Cardiology, Departments of Internal Medicine, National Cheng-Kung University Medical Center, 138 Sheng-Li Road, Tainan 704, Taiwan.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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