rdf:type |
|
lifeskim:mentions |
|
pubmed:issue |
7
|
pubmed:dateCreated |
2006-6-23
|
pubmed:abstractText |
Ca2+-influx through transient receptor potential (TRP) channels was proposed to be important in endothelial function, although the precise role of specific TRP channels is unknown. Here, we investigated the role of the putatively mechanosensitive TRPV4 channel in the mechanisms of endothelium-dependent vasodilatation.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
IM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
Jul
|
pubmed:issn |
1524-4636
|
pubmed:author |
|
pubmed:issnType |
Electronic
|
pubmed:volume |
26
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
1495-502
|
pubmed:dateRevised |
2006-11-15
|
pubmed:meshHeading |
pubmed-meshheading:16675722-Animals,
pubmed-meshheading:16675722-Aorta,
pubmed-meshheading:16675722-Biological Factors,
pubmed-meshheading:16675722-Calcium,
pubmed-meshheading:16675722-Carotid Arteries,
pubmed-meshheading:16675722-Electrophysiology,
pubmed-meshheading:16675722-Endothelial Cells,
pubmed-meshheading:16675722-Endothelium, Vascular,
pubmed-meshheading:16675722-Mechanoreceptors,
pubmed-meshheading:16675722-Nitric Oxide,
pubmed-meshheading:16675722-Patch-Clamp Techniques,
pubmed-meshheading:16675722-Rats,
pubmed-meshheading:16675722-Stress, Mechanical,
pubmed-meshheading:16675722-TRPV Cation Channels,
pubmed-meshheading:16675722-Vasodilation
|
pubmed:year |
2006
|
pubmed:articleTitle |
Evidence for a functional role of endothelial transient receptor potential V4 in shear stress-induced vasodilatation.
|
pubmed:affiliation |
Department of Internal Medicine-Nephrology, Philipps-University, Baldingerstrasse, 35033 Marburg, Germany. rkoehler@med.uni-marburg.de
|
pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|