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rdf:type |
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|
lifeskim:mentions |
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pubmed:issue |
1-2
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pubmed:dateCreated |
1992-3-17
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pubmed:abstractText |
During long-term increase in isoprenaline (pronounced beta-effect) and isoprenaline plus regitine (pure beta-effect) pancreatic insulin-secretion still depended mostly on blood glucose levels. This means that increased beta-effect during normo- or hypoglycemia could not cause a higher insulin-secretion. Only during additional alpha-receptor blockade insulin-secretion was slightly but insufficiently increased. Catecholamines seem to be more regulator than originator of the insulin secretory process.
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pubmed:language |
eng
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pubmed:journal |
|
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pubmed:citationSubset |
IM
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pubmed:chemical |
|
|
pubmed:status |
MEDLINE
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|
pubmed:issn |
0232-1513
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pubmed:author |
|
|
pubmed:issnType |
Print
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pubmed:volume |
43
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
57-61
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pubmed:dateRevised |
2011-11-17
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pubmed:meshHeading |
pubmed-meshheading:1664342-Animals,
pubmed-meshheading:1664342-Blood Glucose,
pubmed-meshheading:1664342-Epinephrine,
pubmed-meshheading:1664342-Insulin,
pubmed-meshheading:1664342-Islets of Langerhans,
pubmed-meshheading:1664342-Isoproterenol,
pubmed-meshheading:1664342-Male,
pubmed-meshheading:1664342-Norepinephrine,
pubmed-meshheading:1664342-Pancreas,
pubmed-meshheading:1664342-Phentolamine,
pubmed-meshheading:1664342-Rats,
pubmed-meshheading:1664342-Rats, Inbred Strains,
pubmed-meshheading:1664342-Receptors, Adrenergic, alpha
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pubmed:year |
1991
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pubmed:articleTitle |
Isoprenaline cannot act on pancreatic beta cells without hyperglycemia or alpha-block.
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pubmed:affiliation |
University of Graz, Institute of Functional Pathology, Austria.
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pubmed:publicationType |
Journal Article
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