pubmed-article:16643366 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:16643366 | lifeskim:mentions | umls-concept:C0034493 | lld:lifeskim |
pubmed-article:16643366 | lifeskim:mentions | umls-concept:C0034144 | lld:lifeskim |
pubmed-article:16643366 | lifeskim:mentions | umls-concept:C0006772 | lld:lifeskim |
pubmed-article:16643366 | lifeskim:mentions | umls-concept:C1864233 | lld:lifeskim |
pubmed-article:16643366 | lifeskim:mentions | umls-concept:C1444783 | lld:lifeskim |
pubmed-article:16643366 | lifeskim:mentions | umls-concept:C0205409 | lld:lifeskim |
pubmed-article:16643366 | lifeskim:mentions | umls-concept:C1413603 | lld:lifeskim |
pubmed-article:16643366 | lifeskim:mentions | umls-concept:C0231491 | lld:lifeskim |
pubmed-article:16643366 | lifeskim:mentions | umls-concept:C0078437 | lld:lifeskim |
pubmed-article:16643366 | lifeskim:mentions | umls-concept:C1292733 | lld:lifeskim |
pubmed-article:16643366 | pubmed:issue | 4 | lld:pubmed |
pubmed-article:16643366 | pubmed:dateCreated | 2006-4-28 | lld:pubmed |
pubmed-article:16643366 | pubmed:abstractText | The occurrence of early afterdepolarizations (EADs) has been related to the incidence of torsades de pointes in drug-induced long QT (LQT). The generation of EADs may be facilitated by Ca(2+)/calmodulin-dependent protein kinase II (CaM kinase). | lld:pubmed |
pubmed-article:16643366 | pubmed:language | eng | lld:pubmed |
pubmed-article:16643366 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16643366 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:16643366 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16643366 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:16643366 | pubmed:month | Apr | lld:pubmed |
pubmed-article:16643366 | pubmed:issn | 1045-3873 | lld:pubmed |
pubmed-article:16643366 | pubmed:author | pubmed-author:LuHua RongHR | lld:pubmed |
pubmed-article:16643366 | pubmed:author | pubmed-author:VlaminckxEddy... | lld:pubmed |
pubmed-article:16643366 | pubmed:author | pubmed-author:GallacherDavi... | lld:pubmed |
pubmed-article:16643366 | pubmed:author | pubmed-author:Van de... | lld:pubmed |
pubmed-article:16643366 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:16643366 | pubmed:volume | 17 | lld:pubmed |
pubmed-article:16643366 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:16643366 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:16643366 | pubmed:pagination | 415-22 | lld:pubmed |
pubmed-article:16643366 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
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pubmed-article:16643366 | pubmed:meshHeading | pubmed-meshheading:16643366... | lld:pubmed |
pubmed-article:16643366 | pubmed:year | 2006 | lld:pubmed |
pubmed-article:16643366 | pubmed:articleTitle | Calmodulin antagonist W-7 prevents sparfloxacin-induced early afterdepolarizations (EADs) in isolated rabbit purkinje fibers: importance of beat-to-beat instability of the repolarization. | lld:pubmed |
pubmed-article:16643366 | pubmed:affiliation | Cardiovascular Safety Research, Johnson & Johnson Pharmaceutical Research & Development, a Division of Janssen Pharmaceutica N.V. B-2340, Beerse, Belgium. hlu@prdbe.jnj.com | lld:pubmed |
pubmed-article:16643366 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:16643366 | pubmed:publicationType | Comparative Study | lld:pubmed |
pubmed-article:16643366 | pubmed:publicationType | In Vitro | lld:pubmed |
pubmed-article:16643366 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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