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rdf:type |
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lifeskim:mentions |
umls-concept:C0008059,
umls-concept:C0017262,
umls-concept:C0020969,
umls-concept:C0185117,
umls-concept:C0332157,
umls-concept:C0439849,
umls-concept:C0445223,
umls-concept:C0557759,
umls-concept:C0597357,
umls-concept:C0678804,
umls-concept:C1325847,
umls-concept:C1552599,
umls-concept:C1704787,
umls-concept:C2911684
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pubmed:issue |
4
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pubmed:dateCreated |
2006-4-24
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pubmed:abstractText |
There is increasing evidence that environmental exposures determining childhood illnesses operate early in life. Prenatal exposure to a farming environment through the mother might also play an important role.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
http://linkedlifedata.com/resource/pubmed/chemical/Antigens, CD14,
http://linkedlifedata.com/resource/pubmed/chemical/RNA, Ribosomal, 18S,
http://linkedlifedata.com/resource/pubmed/chemical/Receptors, Immunologic,
http://linkedlifedata.com/resource/pubmed/chemical/TLR2 protein, human,
http://linkedlifedata.com/resource/pubmed/chemical/TLR4 protein, human,
http://linkedlifedata.com/resource/pubmed/chemical/Toll-Like Receptor 2,
http://linkedlifedata.com/resource/pubmed/chemical/Toll-Like Receptor 4
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pubmed:status |
MEDLINE
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pubmed:month |
Apr
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pubmed:issn |
0091-6749
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pubmed:author |
pubmed-author:BenzMarcus RMR,
pubmed-author:BieliChristianC,
pubmed-author:Braun-FahrländerCharlotteC,
pubmed-author:BrunekreefBertB,
pubmed-author:EgeMarkus JohannesMJ,
pubmed-author:FreiRemoR,
pubmed-author:LauenerRogerR,
pubmed-author:Parsifal Study team,
pubmed-author:PershagenGöranG,
pubmed-author:RiedlerJosefJ,
pubmed-author:ScheyniusAnnikaA,
pubmed-author:Schram-BijkerkDienekeD,
pubmed-author:UblaggerEllenE,
pubmed-author:van HageMarianneM,
pubmed-author:van StrienRobert TheodoorRT,
pubmed-author:von MutiusErikaE
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pubmed:issnType |
Print
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pubmed:volume |
117
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
817-23
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pubmed:dateRevised |
2008-11-21
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pubmed:meshHeading |
pubmed-meshheading:16630939-Adolescent,
pubmed-meshheading:16630939-Animal Husbandry,
pubmed-meshheading:16630939-Animals,
pubmed-meshheading:16630939-Animals, Domestic,
pubmed-meshheading:16630939-Antigens, CD14,
pubmed-meshheading:16630939-Child,
pubmed-meshheading:16630939-Child, Preschool,
pubmed-meshheading:16630939-Cross-Sectional Studies,
pubmed-meshheading:16630939-Europe,
pubmed-meshheading:16630939-Female,
pubmed-meshheading:16630939-Gene Expression,
pubmed-meshheading:16630939-Humans,
pubmed-meshheading:16630939-Hypersensitivity, Immediate,
pubmed-meshheading:16630939-Immunity, Innate,
pubmed-meshheading:16630939-Immunity, Maternally-Acquired,
pubmed-meshheading:16630939-Male,
pubmed-meshheading:16630939-Occupational Exposure,
pubmed-meshheading:16630939-Pregnancy,
pubmed-meshheading:16630939-Prenatal Exposure Delayed Effects,
pubmed-meshheading:16630939-RNA, Ribosomal, 18S,
pubmed-meshheading:16630939-Receptors, Immunologic,
pubmed-meshheading:16630939-Toll-Like Receptor 2,
pubmed-meshheading:16630939-Toll-Like Receptor 4
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pubmed:year |
2006
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pubmed:articleTitle |
Prenatal farm exposure is related to the expression of receptors of the innate immunity and to atopic sensitization in school-age children.
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pubmed:affiliation |
University Children's Hospital Munich, Germany. markus.ege@med.uni-muenchen.de
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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