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rdf:type |
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lifeskim:mentions |
umls-concept:C0003860,
umls-concept:C0004083,
umls-concept:C0012634,
umls-concept:C0017262,
umls-concept:C0185117,
umls-concept:C0205217,
umls-concept:C0221198,
umls-concept:C0441655,
umls-concept:C0546816,
umls-concept:C1337092,
umls-concept:C1522558,
umls-concept:C1705079,
umls-concept:C2911684
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pubmed:issue |
11
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pubmed:dateCreated |
2006-10-19
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pubmed:abstractText |
Patients with giant-cell arteritis (GCA) usually respond dramatically to corticosteroid treatment. However, recurrences are frequent and corticosteroid requirements are highly variable among patients. The aim of our study was to identify genes potentially involved in disease persistence.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
http://linkedlifedata.com/resource/pubmed/chemical/Anti-Inflammatory Agents,
http://linkedlifedata.com/resource/pubmed/chemical/Biological Markers,
http://linkedlifedata.com/resource/pubmed/chemical/CCR2 protein, human,
http://linkedlifedata.com/resource/pubmed/chemical/Chemokine CCL2,
http://linkedlifedata.com/resource/pubmed/chemical/Cytokines,
http://linkedlifedata.com/resource/pubmed/chemical/DNA, Complementary,
http://linkedlifedata.com/resource/pubmed/chemical/Prednisolone,
http://linkedlifedata.com/resource/pubmed/chemical/RNA, Messenger,
http://linkedlifedata.com/resource/pubmed/chemical/Receptors, CCR2,
http://linkedlifedata.com/resource/pubmed/chemical/Receptors, Chemokine
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pubmed:status |
MEDLINE
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pubmed:month |
Nov
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pubmed:issn |
1462-0324
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pubmed:author |
pubmed-author:CidM CMC,
pubmed-author:ElkinMM,
pubmed-author:García-MartínezAA,
pubmed-author:GrauJ MJM,
pubmed-author:Hernández-RodríguezJJ,
pubmed-author:HoffmanM PMP,
pubmed-author:KleinmanH KHK,
pubmed-author:Pla-CampoMM,
pubmed-author:SánchezMM,
pubmed-author:SegarraMM,
pubmed-author:VilardellCC
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pubmed:issnType |
Print
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pubmed:volume |
45
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1356-63
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pubmed:dateRevised |
2007-11-15
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pubmed:meshHeading |
pubmed-meshheading:16621921-Anti-Inflammatory Agents,
pubmed-meshheading:16621921-Biological Markers,
pubmed-meshheading:16621921-Biopsy,
pubmed-meshheading:16621921-Chemokine CCL2,
pubmed-meshheading:16621921-Cytokines,
pubmed-meshheading:16621921-DNA, Complementary,
pubmed-meshheading:16621921-Follow-Up Studies,
pubmed-meshheading:16621921-Gene Expression Regulation,
pubmed-meshheading:16621921-Giant Cell Arteritis,
pubmed-meshheading:16621921-Humans,
pubmed-meshheading:16621921-Prednisolone,
pubmed-meshheading:16621921-Prognosis,
pubmed-meshheading:16621921-Prospective Studies,
pubmed-meshheading:16621921-RNA, Messenger,
pubmed-meshheading:16621921-Receptors, CCR2,
pubmed-meshheading:16621921-Receptors, Chemokine,
pubmed-meshheading:16621921-Recurrence,
pubmed-meshheading:16621921-Temporal Arteries
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pubmed:year |
2006
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pubmed:articleTitle |
Association between increased CCL2 (MCP-1) expression in lesions and persistence of disease activity in giant-cell arteritis.
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pubmed:affiliation |
Department of Internal Medicine, Hospital Clínic, Villarroel 170, Barcelona 08036, Spain. mccid@clinic.ub.es
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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