Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
1
pubmed:dateCreated
2006-7-3
pubmed:abstractText
The effects of prostaglandin E2 (PGE2) are mediated through G-protein coupled receptors, acting via different second messengers. The aim of this study was to characterize the temporal and tissue specific localization and expression of the PGE2 receptor subtypes (EP1-4) in uteroplacental tissues during human pregnancy.
pubmed:grant
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Jul
pubmed:issn
1097-6868
pubmed:author
pubmed:issnType
Electronic
pubmed:volume
195
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
260-9
pubmed:dateRevised
2010-11-18
pubmed:meshHeading
pubmed-meshheading:16615925-Adult, pubmed-meshheading:16615925-Amnion, pubmed-meshheading:16615925-Blotting, Western, pubmed-meshheading:16615925-Chorion, pubmed-meshheading:16615925-Electrophoresis, Polyacrylamide Gel, pubmed-meshheading:16615925-Extraembryonic Membranes, pubmed-meshheading:16615925-Female, pubmed-meshheading:16615925-Humans, pubmed-meshheading:16615925-Immunohistochemistry, pubmed-meshheading:16615925-Labor, Obstetric, pubmed-meshheading:16615925-Placenta, pubmed-meshheading:16615925-Pregnancy, pubmed-meshheading:16615925-Receptors, Prostaglandin E, pubmed-meshheading:16615925-Receptors, Prostaglandin E, EP1 Subtype, pubmed-meshheading:16615925-Receptors, Prostaglandin E, EP2 Subtype, pubmed-meshheading:16615925-Receptors, Prostaglandin E, EP3 Subtype, pubmed-meshheading:16615925-Receptors, Prostaglandin E, EP4 Subtype, pubmed-meshheading:16615925-Trophoblasts
pubmed:year
2006
pubmed:articleTitle
Localization and expression of prostaglandin E2 receptors in human placenta and corresponding fetal membranes with labor.
pubmed:affiliation
Department of Obstetrics and Gynceology, University of Cincinnati, Medical College, Cincinnati, OH 45267, USA. grigsbpl@ucmail.uc.edu
pubmed:publicationType
Journal Article, Research Support, N.I.H., Extramural