pubmed-article:16608032 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:16608032 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
pubmed-article:16608032 | lifeskim:mentions | umls-concept:C0021853 | lld:lifeskim |
pubmed-article:16608032 | lifeskim:mentions | umls-concept:C0205054 | lld:lifeskim |
pubmed-article:16608032 | lifeskim:mentions | umls-concept:C0035126 | lld:lifeskim |
pubmed-article:16608032 | lifeskim:mentions | umls-concept:C0752312 | lld:lifeskim |
pubmed-article:16608032 | lifeskim:mentions | umls-concept:C0700148 | lld:lifeskim |
pubmed-article:16608032 | lifeskim:mentions | umls-concept:C0679823 | lld:lifeskim |
pubmed-article:16608032 | lifeskim:mentions | umls-concept:C0439810 | lld:lifeskim |
pubmed-article:16608032 | pubmed:issue | 68 | lld:pubmed |
pubmed-article:16608032 | pubmed:dateCreated | 2006-4-12 | lld:pubmed |
pubmed-article:16608032 | pubmed:abstractText | The mitogen-activated protein kinase (MAPK) pathways are comprised of key regulatory proteins that control the cellular responses to both proliferation and stress signals. This study was performed to examine the degree of liver damage and MAPK activation induced by ischemia of varying durations, and the association between intestinal congestion and liver dysfunction after hepatic ischemia-reperfusion injury. | lld:pubmed |
pubmed-article:16608032 | pubmed:language | eng | lld:pubmed |
pubmed-article:16608032 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16608032 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:16608032 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16608032 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:16608032 | pubmed:issn | 0172-6390 | lld:pubmed |
pubmed-article:16608032 | pubmed:author | pubmed-author:MorishitaYasu... | lld:pubmed |
pubmed-article:16608032 | pubmed:author | pubmed-author:MatsumotoKosh... | lld:pubmed |
pubmed-article:16608032 | pubmed:author | pubmed-author:TakeyoshiIzum... | lld:pubmed |
pubmed-article:16608032 | pubmed:author | pubmed-author:KobayashiMits... | lld:pubmed |
pubmed-article:16608032 | pubmed:author | pubmed-author:YoshinariDais... | lld:pubmed |
pubmed-article:16608032 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:16608032 | pubmed:volume | 53 | lld:pubmed |
pubmed-article:16608032 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:16608032 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:16608032 | pubmed:pagination | 243-8 | lld:pubmed |
pubmed-article:16608032 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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pubmed-article:16608032 | pubmed:articleTitle | The role of mitogen-activated protein kinases and the participation of intestinal congestion in total hepatic ischemia-reperfusion injury. | lld:pubmed |
pubmed-article:16608032 | pubmed:affiliation | Second Department of Surgery, Gunma University School of Medicine, Maebashi, Japan. | lld:pubmed |
pubmed-article:16608032 | pubmed:publicationType | Journal Article | lld:pubmed |