Source:http://linkedlifedata.com/resource/pubmed/id/16575018
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
1
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pubmed:dateCreated |
2006-6-19
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pubmed:abstractText |
Thixotropy conditioning of inspiratory muscles consisting of maximal inspiratory effort performed at an inflated lung volume is followed by an increase in end-expiratory position of the rib cage in normal human subjects. When performed at a deflated lung volume, conditioning is followed by a reduction in end-expiratory position. The present study was performed to determine whether changes in end-expiratory chest wall and lung volumes occur after thixotropy conditioning. We first examined the acute effects of conditioning on chest wall volume during subsequent five-breath cycles using respiratory inductive plethysmography (n = 8). End-expiratory chest wall volume increased after conditioning at an inflated lung volume (P < 0.05), which was attained mainly by rib cage movements. Conditioning at a deflated lung volume was followed by reductions in end-expiratory chest wall volume, which was explained by rib cage and abdominal volume changes (P < 0.05). End-expiratory esophageal pressure decreased and increased after conditioning at inflated and deflated lung volumes, respectively (n = 3). These changes in end-expiratory volumes and esophageal pressure were greatest for the first breath after conditioning. We also found that an increase in spirometrically determined inspiratory capacity (n = 13) was maintained for 3 min after conditioning at a deflated lung volume, and a decrease for 1 min after conditioning at an inflated lung volume. Helium-dilution end-expiratory lung volume increased and decreased after conditioning at inflated and deflated lung volumes, respectively (both P < 0.05; n = 11). These results suggest that thixotropy conditioning changes end-expiratory volume of the chest wall and lung in normal human subjects.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Jul
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pubmed:issn |
8750-7587
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
101
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
298-306
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:16575018-Adult,
pubmed-meshheading:16575018-Elasticity,
pubmed-meshheading:16575018-Exhalation,
pubmed-meshheading:16575018-Forced Expiratory Volume,
pubmed-meshheading:16575018-Functional Residual Capacity,
pubmed-meshheading:16575018-Helium,
pubmed-meshheading:16575018-Humans,
pubmed-meshheading:16575018-Inhalation,
pubmed-meshheading:16575018-Lung,
pubmed-meshheading:16575018-Lung Volume Measurements,
pubmed-meshheading:16575018-Male,
pubmed-meshheading:16575018-Respiratory Muscles,
pubmed-meshheading:16575018-Thoracic Wall,
pubmed-meshheading:16575018-Total Lung Capacity,
pubmed-meshheading:16575018-Vital Capacity
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pubmed:year |
2006
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pubmed:articleTitle |
Acute effects of thixotropy conditioning of inspiratory muscles on end-expiratory chest wall and lung volumes in normal humans.
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pubmed:affiliation |
Department of Physiology, Showa University School of Medicine 1-5-8 Hatanodai, Shinagawa-ku 142-8555, Tokyo, Japan. masahiko@med.showa-u.ac.jp
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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