16574899

Source:http://linkedlifedata.com/resource/pubmed/id/16574899

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0003842, umls-concept:C0018270, umls-concept:C0020443, umls-concept:C0020456, umls-concept:C0021655, umls-concept:C0026809, umls-concept:C0392756, umls-concept:C1948058
pubmed:issue	6
pubmed:dateCreated	2006-5-19
pubmed:abstractText	Collateral artery development (arteriogenesis), a vital compensatory mechanism in patients with arterial obstructive disease, may be deregulated by vascular risk factors, eg, diabetes or hypercholesterolemia. Here, we compared the effects of either disturbed glucose metabolism or disturbed lipid metabolism on arteriogenesis.
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/9505803
pubmed:citationSubset	IM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/Apolipoprotein E3, http://linkedlifedata.com/resource/pubmed/chemical/Apolipoproteins E, http://linkedlifedata.com/resource/pubmed/chemical/Blood Glucose, http://linkedlifedata.com/resource/pubmed/chemical/Cholesterol, http://linkedlifedata.com/resource/pubmed/chemical/Insulin, http://linkedlifedata.com/resource/pubmed/chemical/Lipids, http://linkedlifedata.com/resource/pubmed/chemical/apolipoprotein E3 (Leidein)
pubmed:status	MEDLINE
pubmed:month	Jun
pubmed:issn	1524-4636
pubmed:author	pubmed-author:EilersPaul H CPH, pubmed-author:QuaxPaul H APH, pubmed-author:VerloopRobert ERE, pubmed-author:VosholPeter JPJ, pubmed-author:de VriesMargreetM, pubmed-author:van BockelJ HajoJH, pubmed-author:van HinsberghVictor W MVW, pubmed-author:van WeelVincentV
pubmed:issnType	Electronic
pubmed:volume	26
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	1383-90
pubmed:dateRevised	2011-11-17
pubmed:meshHeading	pubmed-meshheading:16574899-Acute Disease, pubmed-meshheading:16574899-Animals, pubmed-meshheading:16574899-Apolipoprotein E3, pubmed-meshheading:16574899-Apolipoproteins E, pubmed-meshheading:16574899-Arteries, pubmed-meshheading:16574899-Blood Glucose, pubmed-meshheading:16574899-Cholesterol, pubmed-meshheading:16574899-Collateral Circulation, pubmed-meshheading:16574899-Diabetes Mellitus, Experimental, pubmed-meshheading:16574899-Hindlimb, pubmed-meshheading:16574899-Hypercholesterolemia, pubmed-meshheading:16574899-Hyperglycemia, pubmed-meshheading:16574899-Hyperlipidemias, pubmed-meshheading:16574899-Insulin, pubmed-meshheading:16574899-Insulin Resistance, pubmed-meshheading:16574899-Ischemia, pubmed-meshheading:16574899-Lipids, pubmed-meshheading:16574899-Male, pubmed-meshheading:16574899-Mice, pubmed-meshheading:16574899-Mice, Inbred Strains, pubmed-meshheading:16574899-Neovascularization, Physiologic
pubmed:year	2006
pubmed:articleTitle	Hypercholesterolemia reduces collateral artery growth more dominantly than hyperglycemia or insulin resistance in mice.
pubmed:affiliation	Gaubius Laboratory TNO Biomedical Research, Leiden, The Netherlands.
pubmed:publicationType	Journal Article, Comparative Study, Research Support, Non-U.S. Gov't