Source:http://linkedlifedata.com/resource/pubmed/id/16545652
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
6
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pubmed:dateCreated |
2006-3-20
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pubmed:abstractText |
The hypothesis that an increased or prolonged Ca2+ transient during an abbreviated action potential can give rise to early afterdepolarizations (EADs) and triggered arrhythmia by enhanced forward sodium-calcium (Na-Ca) exchange was examined.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
AIM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Mar
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pubmed:issn |
1558-3597
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pubmed:author | |
pubmed:issnType |
Electronic
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pubmed:day |
21
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pubmed:volume |
47
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1196-206
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pubmed:dateRevised |
2007-11-15
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pubmed:meshHeading |
pubmed-meshheading:16545652-Action Potentials,
pubmed-meshheading:16545652-Animals,
pubmed-meshheading:16545652-Arrhythmias, Cardiac,
pubmed-meshheading:16545652-Calcium,
pubmed-meshheading:16545652-Dogs,
pubmed-meshheading:16545652-Male,
pubmed-meshheading:16545652-Pulmonary Veins,
pubmed-meshheading:16545652-Sodium-Calcium Exchanger
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pubmed:year |
2006
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pubmed:articleTitle |
Sodium-calcium exchange initiated by the Ca2+ transient: an arrhythmia trigger within pulmonary veins.
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pubmed:affiliation |
University of Oklahoma Health Sciences Center and the DVA Medical Center, Oklahoma City, Oklahoma, USA. eugene-patterson@ouhsc.edu
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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