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rdf:type |
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lifeskim:mentions |
umls-concept:C0018801,
umls-concept:C0026809,
umls-concept:C0202042,
umls-concept:C0242606,
umls-concept:C0439834,
umls-concept:C0449258,
umls-concept:C0599946,
umls-concept:C1299007,
umls-concept:C2267008,
umls-concept:C2587213,
umls-concept:C2756986
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pubmed:issue |
1
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pubmed:dateCreated |
2006-3-27
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pubmed:abstractText |
It has been suggested that reduction in glucose levels contributes to the prolongation of life span of rodents in conjunction with restricted food intake, and hyperglycemia has been confirmed as a risk factor for cardiovascular disease (CVD), raising the possibility that better glycemic control could slow the progression of CVD. This study was designed to determine whether impaired glucose tolerance develops during the progression of cardiac hypertrophy and heart failure, and whether tight glycemic control could reduce the severity of heart failure.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
http://linkedlifedata.com/resource/pubmed/chemical/Acetophenones,
http://linkedlifedata.com/resource/pubmed/chemical/Fatty Acids, Nonesterified,
http://linkedlifedata.com/resource/pubmed/chemical/Glucose,
http://linkedlifedata.com/resource/pubmed/chemical/Inositol,
http://linkedlifedata.com/resource/pubmed/chemical/Insulin,
http://linkedlifedata.com/resource/pubmed/chemical/NADPH Oxidase,
http://linkedlifedata.com/resource/pubmed/chemical/acetovanillone,
http://linkedlifedata.com/resource/pubmed/chemical/alpha-Glucosidases,
http://linkedlifedata.com/resource/pubmed/chemical/neutrophil cytosolic factor 1,
http://linkedlifedata.com/resource/pubmed/chemical/voglibose
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pubmed:status |
MEDLINE
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pubmed:month |
Apr
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pubmed:issn |
0008-6363
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:day |
1
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pubmed:volume |
70
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
107-16
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pubmed:dateRevised |
2011-11-17
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pubmed:meshHeading |
pubmed-meshheading:16510136-Acetophenones,
pubmed-meshheading:16510136-Animals,
pubmed-meshheading:16510136-Blotting, Western,
pubmed-meshheading:16510136-Body Weight,
pubmed-meshheading:16510136-Cells, Cultured,
pubmed-meshheading:16510136-Disease Progression,
pubmed-meshheading:16510136-Echocardiography,
pubmed-meshheading:16510136-Fatty Acids, Nonesterified,
pubmed-meshheading:16510136-Glucose,
pubmed-meshheading:16510136-Heart Failure,
pubmed-meshheading:16510136-Hyperglycemia,
pubmed-meshheading:16510136-Inositol,
pubmed-meshheading:16510136-Insulin,
pubmed-meshheading:16510136-Lung,
pubmed-meshheading:16510136-Male,
pubmed-meshheading:16510136-Mice,
pubmed-meshheading:16510136-Mice, Inbred C57BL,
pubmed-meshheading:16510136-Models, Animal,
pubmed-meshheading:16510136-Myocardium,
pubmed-meshheading:16510136-Myocytes, Cardiac,
pubmed-meshheading:16510136-NADPH Oxidase,
pubmed-meshheading:16510136-Organ Size,
pubmed-meshheading:16510136-Oxidative Stress,
pubmed-meshheading:16510136-Rats,
pubmed-meshheading:16510136-Reverse Transcriptase Polymerase Chain Reaction,
pubmed-meshheading:16510136-Ventricular Dysfunction, Left,
pubmed-meshheading:16510136-alpha-Glucosidases
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pubmed:year |
2006
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pubmed:articleTitle |
Control of plasma glucose with alpha-glucosidase inhibitor attenuates oxidative stress and slows the progression of heart failure in mice.
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pubmed:affiliation |
Department of Cardiovascular Medicine, Osaka University Graduate School of Medicine, 2-2 Yamadaoka, Suita, Osaka 565-0871, Japan.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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