Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
5
pubmed:dateCreated
2006-3-1
pubmed:abstractText
Microscopic human cancers can remain dormant for life. Tumor progression depends on sequential events, including a switch to the angiogenic phenotype, i.e., initial recruitment of new vessels. We previously demonstrated that human tumors contain tumor cell populations that are heterogeneous in angiogenic activity. Here, we separated angiogenic from nonangiogenic human tumor cell populations and compared their growth.
pubmed:commentsCorrections
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Mar
pubmed:issn
1460-2105
pubmed:author
pubmed:issnType
Electronic
pubmed:day
1
pubmed:volume
98
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
316-25
pubmed:dateRevised
2007-3-13
pubmed:meshHeading
pubmed-meshheading:16507828-Adenocarcinoma, pubmed-meshheading:16507828-Animals, pubmed-meshheading:16507828-Cell Line, Tumor, pubmed-meshheading:16507828-Cell Proliferation, pubmed-meshheading:16507828-Enzyme-Linked Immunosorbent Assay, pubmed-meshheading:16507828-Fibroblast Growth Factor 2, pubmed-meshheading:16507828-Gene Expression Regulation, Neoplastic, pubmed-meshheading:16507828-Glioblastoma, pubmed-meshheading:16507828-Humans, pubmed-meshheading:16507828-Immunoblotting, pubmed-meshheading:16507828-Immunohistochemistry, pubmed-meshheading:16507828-In Situ Nick-End Labeling, pubmed-meshheading:16507828-Mice, pubmed-meshheading:16507828-Mice, SCID, pubmed-meshheading:16507828-Neovascularization, Pathologic, pubmed-meshheading:16507828-Osteosarcoma, pubmed-meshheading:16507828-Phenotype, pubmed-meshheading:16507828-Thrombospondin 1, pubmed-meshheading:16507828-Time Factors, pubmed-meshheading:16507828-Tumor Cells, Cultured, pubmed-meshheading:16507828-Vascular Endothelial Growth Factor A
pubmed:year
2006
pubmed:articleTitle
A model of human tumor dormancy: an angiogenic switch from the nonangiogenic phenotype.
pubmed:affiliation
Department of Surgery, Children's Hospital, Harvard Medical School, Boston, MA, USA.
pubmed:publicationType
Journal Article, Research Support, U.S. Gov't, Non-P.H.S., Research Support, Non-U.S. Gov't