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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
3
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pubmed:dateCreated |
2006-2-23
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pubmed:abstractText |
Several studies have indicated the involvement of nitric oxide (NO) in the pathogenesis of tardive dyskinesia (TD), an incapacitating adverse movement disorder associated with long-term antipsychotic treatment. In human brain, the NO could be generated by endothelial nitric oxide synthase (NOS3). In this study, we studied whether the genetic variants in human NOS3 gene is associated with TD in patients with schizophrenia.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Mar
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pubmed:issn |
1744-6872
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
16
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
151-7
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pubmed:dateRevised |
2008-11-21
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pubmed:meshHeading |
pubmed-meshheading:16495774-Adult,
pubmed-meshheading:16495774-Alleles,
pubmed-meshheading:16495774-Antipsychotic Agents,
pubmed-meshheading:16495774-Case-Control Studies,
pubmed-meshheading:16495774-Dyskinesia, Drug-Induced,
pubmed-meshheading:16495774-Genetic Variation,
pubmed-meshheading:16495774-Haplotypes,
pubmed-meshheading:16495774-Humans,
pubmed-meshheading:16495774-Middle Aged,
pubmed-meshheading:16495774-Nitric Oxide Synthase Type III,
pubmed-meshheading:16495774-Odds Ratio,
pubmed-meshheading:16495774-Pharmacogenetics,
pubmed-meshheading:16495774-Promoter Regions, Genetic,
pubmed-meshheading:16495774-Schizophrenia
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pubmed:year |
2006
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pubmed:articleTitle |
Haplotype analysis of endothelial nitric oxide synthase (NOS3) genetic variants and tardive dyskinesia in patients with schizophrenia.
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pubmed:affiliation |
Institute of Clinical Medicine, Yuli Veterans Hospital, Yuli, Hualien, Taiwan, ROC.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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