| pubmed-article:1649403 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:1649403 | lifeskim:mentions | umls-concept:C0006104 | lld:lifeskim |
| pubmed-article:1649403 | lifeskim:mentions | umls-concept:C0086045 | lld:lifeskim |
| pubmed-article:1649403 | lifeskim:mentions | umls-concept:C0162557 | lld:lifeskim |
| pubmed-article:1649403 | lifeskim:mentions | umls-concept:C0205250 | lld:lifeskim |
| pubmed-article:1649403 | pubmed:issue | 7 | lld:pubmed |
| pubmed-article:1649403 | pubmed:dateCreated | 1991-8-16 | lld:pubmed |
| pubmed-article:1649403 | pubmed:abstractText | Increased gamma-aminobutyric acid (GABA) neurotransmission has been implicated in the pathogenesis of hepatic encephalopathy. The mechanism by which GABA-ergic activity is increased in hepatic failure is unclear, but recent studies in animals with encephalopathy due to fulminant hepatic failure suggest that GABA-ergic neurotransmission may be increased by the presence of elevated concentrations of benzodiazepine agonists such as diazepam and N-desmethyldiazepam. | lld:pubmed |
| pubmed-article:1649403 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1649403 | pubmed:language | eng | lld:pubmed |
| pubmed-article:1649403 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1649403 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:1649403 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:1649403 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:1649403 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:1649403 | pubmed:issn | 0028-4793 | lld:pubmed |
| pubmed-article:1649403 | pubmed:author | pubmed-author:SkolnickPP | lld:pubmed |
| pubmed-article:1649403 | pubmed:author | pubmed-author:MurataYY | lld:pubmed |
| pubmed-article:1649403 | pubmed:author | pubmed-author:WilliamsRR | lld:pubmed |
| pubmed-article:1649403 | pubmed:author | pubmed-author:JonesE AEA | lld:pubmed |
| pubmed-article:1649403 | pubmed:author | pubmed-author:HughesR DRD | lld:pubmed |
| pubmed-article:1649403 | pubmed:author | pubmed-author:HarrisonP MPM | lld:pubmed |
| pubmed-article:1649403 | pubmed:author | pubmed-author:BasiliA GAG | lld:pubmed |
| pubmed-article:1649403 | pubmed:author | pubmed-author:PannellLL | lld:pubmed |
| pubmed-article:1649403 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:1649403 | pubmed:day | 15 | lld:pubmed |
| pubmed-article:1649403 | pubmed:volume | 325 | lld:pubmed |
| pubmed-article:1649403 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:1649403 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:1649403 | pubmed:pagination | 473-8 | lld:pubmed |
| pubmed-article:1649403 | pubmed:dateRevised | 2010-3-24 | lld:pubmed |
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| pubmed-article:1649403 | pubmed:meshHeading | pubmed-meshheading:1649403-... | lld:pubmed |
| pubmed-article:1649403 | pubmed:year | 1991 | lld:pubmed |
| pubmed-article:1649403 | pubmed:articleTitle | Elevated brain concentrations of 1,4-benzodiazepines in fulminant hepatic failure. | lld:pubmed |
| pubmed-article:1649403 | pubmed:affiliation | Laboratory of Neuroscience, National Institute of Diabetes and Digestive and Kidney Diseases, National Institutes of Health, Bethesda, MD 20892. | lld:pubmed |
| pubmed-article:1649403 | pubmed:publicationType | Journal Article | lld:pubmed |
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