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| Predicate | Object |
|---|---|
| rdf:type | |
| lifeskim:mentions | |
| pubmed:dateCreated |
1991-8-14
|
| pubmed:abstractText |
An uncontrolled and sustained increase in cytosolic Ca2+ concentration has been implicated as an early event in the development of anoxic or toxic cell injury. More recently it has become clear that an elevation of cytosolic Ca2+ is also involved in programmed cell death in the immune system. Here, we review some of our recent studies and provide further evidence for the role of Ca2+ in cell killing.
|
| pubmed:language |
eng
|
| pubmed:journal | |
| pubmed:citationSubset |
IM
|
| pubmed:chemical | |
| pubmed:status |
MEDLINE
|
| pubmed:issn |
0065-2598
|
| pubmed:author | |
| pubmed:issnType |
Print
|
| pubmed:volume |
283
|
| pubmed:owner |
NLM
|
| pubmed:authorsComplete |
Y
|
| pubmed:pagination |
419-25
|
| pubmed:dateRevised |
2005-11-16
|
| pubmed:meshHeading |
pubmed-meshheading:1648868-Animals,
pubmed-meshheading:1648868-Calcium,
pubmed-meshheading:1648868-Cell Survival,
pubmed-meshheading:1648868-Cytoskeleton,
pubmed-meshheading:1648868-Cytosol,
pubmed-meshheading:1648868-Endonucleases,
pubmed-meshheading:1648868-Endopeptidases,
pubmed-meshheading:1648868-Homeostasis,
pubmed-meshheading:1648868-Phospholipases
|
| pubmed:year |
1991
|
| pubmed:articleTitle |
Role of calcium in toxic and programmed cell death.
|
| pubmed:affiliation |
Department of Toxicology, Karolinska Institutet, Stockholm, Sweden.
|
| pubmed:publicationType |
Journal Article,
Review
|