rdf:type |
|
lifeskim:mentions |
umls-concept:C0014442,
umls-concept:C0019010,
umls-concept:C0023884,
umls-concept:C0034693,
umls-concept:C0034721,
umls-concept:C0332298,
umls-concept:C0391871,
umls-concept:C0392747,
umls-concept:C0521451,
umls-concept:C0728938,
umls-concept:C1096176,
umls-concept:C1546988
|
pubmed:issue |
2
|
pubmed:dateCreated |
2006-2-17
|
pubmed:abstractText |
Serum activities of assumed organ-specific enzymes are useful protein markers in the diagnosis of necrotic liver diseases. However, after partial hepatectomy (PH) in rats, remaining hepatocytes proliferate to restore the lost liver mass, even when there is a drastic but selective elevation of serum enzyme activities. The aim of the present study was to elucidate the underlying mechanisms involved in this PH-induced enhancement of enzyme release.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
IM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
Feb
|
pubmed:issn |
0036-5521
|
pubmed:author |
|
pubmed:issnType |
Print
|
pubmed:volume |
41
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
223-33
|
pubmed:dateRevised |
2006-11-15
|
pubmed:meshHeading |
pubmed-meshheading:16484128-Alanine Transaminase,
pubmed-meshheading:16484128-Animals,
pubmed-meshheading:16484128-Aspartate Aminotransferase, Mitochondrial,
pubmed-meshheading:16484128-Biological Markers,
pubmed-meshheading:16484128-Blood Flow Velocity,
pubmed-meshheading:16484128-Disease Models, Animal,
pubmed-meshheading:16484128-Hepatectomy,
pubmed-meshheading:16484128-L-Lactate Dehydrogenase,
pubmed-meshheading:16484128-Liver Circulation,
pubmed-meshheading:16484128-Liver Diseases,
pubmed-meshheading:16484128-Liver Regeneration,
pubmed-meshheading:16484128-Male,
pubmed-meshheading:16484128-Mitochondria, Liver,
pubmed-meshheading:16484128-Rats,
pubmed-meshheading:16484128-Rats, Wistar,
pubmed-meshheading:16484128-Reperfusion Injury
|
pubmed:year |
2006
|
pubmed:articleTitle |
Predominance of released mitochondrial enzymes by partial hepatectomy-induced rat regenerating liver is controlled by hemodynamic changes and not related to mitochondrial damage.
|
pubmed:affiliation |
Departamento de Biología Celular, Instituto de Fisiología Celular, Universidad Nacional Autónoma de México (UNAM), Mexico.
|
pubmed:publicationType |
Journal Article,
Comparative Study,
Research Support, Non-U.S. Gov't
|