Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
4
pubmed:dateCreated
2006-3-24
pubmed:abstractText
This study characterized the role of insulin receptors and resistance on vascular endothelial growth factor (VEGF) expression and myocardial vascularization in physiological conditions and after ischemia.
pubmed:grant
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Apr
pubmed:issn
1524-4636
pubmed:author
pubmed:issnType
Electronic
pubmed:volume
26
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
787-93
pubmed:dateRevised
2011-11-17
pubmed:meshHeading
pubmed-meshheading:16469952-Androstadienes, pubmed-meshheading:16469952-Animals, pubmed-meshheading:16469952-Cells, Cultured, pubmed-meshheading:16469952-Coronary Vessels, pubmed-meshheading:16469952-Flavonoids, pubmed-meshheading:16469952-Humans, pubmed-meshheading:16469952-Insulin, pubmed-meshheading:16469952-Insulin Resistance, pubmed-meshheading:16469952-Male, pubmed-meshheading:16469952-Mice, pubmed-meshheading:16469952-Myocardial Ischemia, pubmed-meshheading:16469952-Myocytes, Cardiac, pubmed-meshheading:16469952-Neovascularization, Pathologic, pubmed-meshheading:16469952-Phosphatidylinositol 3-Kinases, pubmed-meshheading:16469952-Protein Kinase Inhibitors, pubmed-meshheading:16469952-Proto-Oncogene Proteins c-akt, pubmed-meshheading:16469952-Rats, pubmed-meshheading:16469952-Rats, Zucker, pubmed-meshheading:16469952-Receptor, Insulin, pubmed-meshheading:16469952-Signal Transduction, pubmed-meshheading:16469952-Vascular Endothelial Growth Factor A
pubmed:year
2006
pubmed:articleTitle
Regulation of vascular endothelial growth factor expression and vascularization in the myocardium by insulin receptor and PI3K/Akt pathways in insulin resistance and ischemia.
pubmed:affiliation
Research Division, Joslin Diabetes Center, Harvard Medical School, Boston, MA, USA.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't, Research Support, N.I.H., Extramural