16373645

Source:http://linkedlifedata.com/resource/pubmed/id/16373645

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0017262, umls-concept:C0185117, umls-concept:C1366911, umls-concept:C1427709, umls-concept:C2348042, umls-concept:C2911684
pubmed:issue	2
pubmed:dateCreated	2006-1-24
pubmed:abstractText	Mutations in CCM2 (MGC4607 or malcavernin) cause familial cerebral cavernous malformation (CCM), an autosomal dominant neurovascular disease. Both the function of this molecule and the pathogenesis of the disease remain elusive.
pubmed:grant	http://linkedlifedata.com/resource/pubmed/grant/R01NS046521
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/0235266
pubmed:citationSubset	IM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/CCM2 protein, human, http://linkedlifedata.com/resource/pubmed/chemical/Carrier Proteins, http://linkedlifedata.com/resource/pubmed/chemical/Ccm1 protein, mouse, http://linkedlifedata.com/resource/pubmed/chemical/KRIT1 protein, human, http://linkedlifedata.com/resource/pubmed/chemical/Microtubule-Associated Proteins, http://linkedlifedata.com/resource/pubmed/chemical/Proto-Oncogene Proteins, http://linkedlifedata.com/resource/pubmed/chemical/RNA, Messenger
pubmed:status	MEDLINE
pubmed:month	Feb
pubmed:issn	1524-4628
pubmed:author	pubmed-author:GucluBulentB, pubmed-author:GunelMuratM, pubmed-author:LouviAngelikiA, pubmed-author:OzturkAli KAK, pubmed-author:PricolaKatie LKL, pubmed-author:SekerAskinA
pubmed:issnType	Electronic
pubmed:volume	37
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	518-23
pubmed:dateRevised	2008-5-6
pubmed:meshHeading	pubmed-meshheading:16373645-Animals, pubmed-meshheading:16373645-Blotting, Western, pubmed-meshheading:16373645-Brain, pubmed-meshheading:16373645-COS Cells, pubmed-meshheading:16373645-Carrier Proteins, pubmed-meshheading:16373645-Cells, Cultured, pubmed-meshheading:16373645-Central Nervous System, pubmed-meshheading:16373645-Cercopithecus aethiops, pubmed-meshheading:16373645-Cerebral Cortex, pubmed-meshheading:16373645-Endothelium, Vascular, pubmed-meshheading:16373645-Humans, pubmed-meshheading:16373645-Immunohistochemistry, pubmed-meshheading:16373645-In Situ Hybridization, pubmed-meshheading:16373645-Mice, pubmed-meshheading:16373645-Microtubule-Associated Proteins, pubmed-meshheading:16373645-Muscle, Smooth, pubmed-meshheading:16373645-Mutation, pubmed-meshheading:16373645-Neurons, pubmed-meshheading:16373645-Phenotype, pubmed-meshheading:16373645-Proto-Oncogene Proteins, pubmed-meshheading:16373645-RNA, Messenger, pubmed-meshheading:16373645-Signal Transduction, pubmed-meshheading:16373645-Time Factors, pubmed-meshheading:16373645-Two-Hybrid System Techniques, pubmed-meshheading:16373645-Umbilical Veins
pubmed:year	2006
pubmed:articleTitle	CCM2 expression parallels that of CCM1.
pubmed:affiliation	Department of Neurosurgery, Yale University School of Medicine, New Haven, CT 06510, USA.
pubmed:publicationType	Journal Article, Research Support, N.I.H., Extramural