rdf:type |
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lifeskim:mentions |
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pubmed:issue |
24 Pt 1
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pubmed:dateCreated |
2005-12-19
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pubmed:abstractText |
The Polycomb group gene, EZH2, functions as a transcriptional repressor involved in gene silencing. Amplification of EZH2 has been reported in several malignancies, including prostate, breast, and lymphoma. We evaluated EZH2 mRNA and protein expression in bladder specimens from patients and the EZH2 mRNA expression in five bladder cancer cell lines.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
|
pubmed:status |
MEDLINE
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pubmed:month |
Dec
|
pubmed:issn |
1078-0432
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pubmed:author |
|
pubmed:issnType |
Print
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pubmed:day |
15
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pubmed:volume |
11
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pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
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pubmed:pagination |
8570-6
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pubmed:dateRevised |
2008-11-21
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pubmed:meshHeading |
pubmed-meshheading:16361539-Adult,
pubmed-meshheading:16361539-Aged,
pubmed-meshheading:16361539-Aged, 80 and over,
pubmed-meshheading:16361539-Carcinoma, Transitional Cell,
pubmed-meshheading:16361539-Cell Line, Tumor,
pubmed-meshheading:16361539-DNA-Binding Proteins,
pubmed-meshheading:16361539-Female,
pubmed-meshheading:16361539-Gene Expression Regulation, Neoplastic,
pubmed-meshheading:16361539-Humans,
pubmed-meshheading:16361539-Male,
pubmed-meshheading:16361539-Middle Aged,
pubmed-meshheading:16361539-RNA, Messenger,
pubmed-meshheading:16361539-RNA, Neoplasm,
pubmed-meshheading:16361539-Transcription, Genetic,
pubmed-meshheading:16361539-Transcription Factors,
pubmed-meshheading:16361539-Transcriptional Activation,
pubmed-meshheading:16361539-Urinary Bladder,
pubmed-meshheading:16361539-Urinary Bladder Neoplasms
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pubmed:year |
2005
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pubmed:articleTitle |
Increased expression of the polycomb group gene, EZH2, in transitional cell carcinoma of the bladder.
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pubmed:affiliation |
Department of Urology, The New York Presbyterian Hospital, Weill Medical College of Cornell University, New York, New York 10021, USA.
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pubmed:publicationType |
Journal Article,
Comparative Study,
Research Support, Non-U.S. Gov't,
Research Support, N.I.H., Extramural
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