Source:http://linkedlifedata.com/resource/pubmed/id/16340751
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
1
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pubmed:dateCreated |
2005-12-12
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pubmed:abstractText |
Overexpression of epidermal growth factor (EGF) and EGF receptor has been associated with progression and invasive phenotype in pancreatic cancer. However, the underlying molecular mechanism by which EGF worked in pancreatic cancer cells has been poorly understood. In this study, we determined the effect of EGF on the invasiveness and the related regulatory mechanism in two pancreatic cancer cell lines NOR-P1 and KP4.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Jan
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pubmed:issn |
1536-4828
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pubmed:author | |
pubmed:issnType |
Electronic
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pubmed:volume |
32
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
101-9
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pubmed:meshHeading |
pubmed-meshheading:16340751-Cell Adhesion,
pubmed-meshheading:16340751-Cell Division,
pubmed-meshheading:16340751-Cell Line, Tumor,
pubmed-meshheading:16340751-Epidermal Growth Factor,
pubmed-meshheading:16340751-Humans,
pubmed-meshheading:16340751-Matrix Metalloproteinases,
pubmed-meshheading:16340751-NF-kappa B,
pubmed-meshheading:16340751-Neoplasm Invasiveness,
pubmed-meshheading:16340751-Pancreatic Neoplasms,
pubmed-meshheading:16340751-Peptide Hydrolases
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pubmed:year |
2006
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pubmed:articleTitle |
Epidermal growth factor promotes invasiveness of pancreatic cancer cells through NF-kappaB-mediated proteinase productions.
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pubmed:affiliation |
Department of Surgery, The First Affiliated Hospital of China Medical University, Shenyang, China. haozhang@mail.cmu.edu.cn
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pubmed:publicationType |
Journal Article
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