Linked Life Data

16320083

Source:http://linkedlifedata.com/resource/pubmed/id/16320083

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
12
pubmed:dateCreated
2005-12-1
pubmed:abstractText
Gestational diabetes mellitus (GDM) and type 2 diabetes share a common pathophysiological background, including beta cell dysfunction and insulin resistance. In addition, women with GDM are at increased risk of developing type 2 diabetes later in life. Our aim was to investigate whether, like type 2 diabetes, GDM has a genetic predisposition by studying five common polymorphisms in four candidate genes that have previously been associated with type 2 diabetes.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
http://linkedlifedata.com/resource/pubmed/chemical/Calpain, http://linkedlifedata.com/resource/pubmed/chemical/IRS1 protein, human, http://linkedlifedata.com/resource/pubmed/chemical/Insulin Receptor Substrate Proteins, http://linkedlifedata.com/resource/pubmed/chemical/Ion Channels, http://linkedlifedata.com/resource/pubmed/chemical/Kir6.2 channel, http://linkedlifedata.com/resource/pubmed/chemical/Membrane Transport Proteins, http://linkedlifedata.com/resource/pubmed/chemical/Mitochondrial Proteins, http://linkedlifedata.com/resource/pubmed/chemical/Phosphoproteins, http://linkedlifedata.com/resource/pubmed/chemical/Potassium Channels, Inwardly..., http://linkedlifedata.com/resource/pubmed/chemical/calpain 10, http://linkedlifedata.com/resource/pubmed/chemical/mitochondrial uncoupling protein 2
pubmed:status
MEDLINE
pubmed:month
Dec
pubmed:issn
0012-186X
pubmed:author
pubmed:issnType
Print
pubmed:volume
48
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
2544-51
pubmed:dateRevised
2008-11-21
pubmed:meshHeading
pubmed-meshheading:16320083-Adult, pubmed-meshheading:16320083-Alleles, pubmed-meshheading:16320083-Calpain, pubmed-meshheading:16320083-Case-Control Studies, pubmed-meshheading:16320083-Diabetes, Gestational, pubmed-meshheading:16320083-Diabetes Mellitus, Type 2, pubmed-meshheading:16320083-Female, pubmed-meshheading:16320083-Gene Frequency, pubmed-meshheading:16320083-Genetic Predisposition to Disease, pubmed-meshheading:16320083-Genotype, pubmed-meshheading:16320083-Humans, pubmed-meshheading:16320083-Insulin Receptor Substrate Proteins, pubmed-meshheading:16320083-Insulin Resistance, pubmed-meshheading:16320083-Insulin-Secreting Cells, pubmed-meshheading:16320083-Ion Channels, pubmed-meshheading:16320083-Membrane Transport Proteins, pubmed-meshheading:16320083-Mitochondrial Proteins, pubmed-meshheading:16320083-Odds Ratio, pubmed-meshheading:16320083-Phosphoproteins, pubmed-meshheading:16320083-Polymorphism, Genetic, pubmed-meshheading:16320083-Potassium Channels, Inwardly Rectifying, pubmed-meshheading:16320083-Pregnancy, pubmed-meshheading:16320083-Risk, pubmed-meshheading:16320083-Sweden
pubmed:year
2005
pubmed:articleTitle
Association of the E23K polymorphism in the KCNJ11 gene with gestational diabetes mellitus.
pubmed:affiliation
Department of Clinical Sciences/Diabetes and Endocrinology, Malmö University Hospital, Lund University, Malmö, Sweden. nael.shaat@med.lu.se
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't