Linked Life Data

16300275

Source:http://linkedlifedata.com/resource/pubmed/id/16300275

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:dateCreated
2005-11-22
pubmed:abstractText
Encapsulating peritoneal sclerosis (EPS) is one of the most serious complications of long-term peritoneal dialysis (PD). Long-term PD therapy has been suggested as a risk factor for EPS development among patients in Japan and Australia. Although the primary mechanism of EPS development has not been clarified, histologic changes of the peritoneum associated with prolonged PD are postulated to be causally related. The present article proposes a working hypothesis--the plasma leak-to-response hypothesis--that vascular alterations in the peritoneum of long-term PD patients play a crucial role in the initiation and development of EPS.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:status
MEDLINE
pubmed:month
Apr
pubmed:issn
0896-8608
pubmed:author
pubmed:issnType
Print
pubmed:volume
25 Suppl 4
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
S71-6
pubmed:meshHeading
pubmed:year
2005
pubmed:articleTitle
The plasma leak-to-response hypothesis: a working hypothesis on the pathogenesis of encapsulating peritoneal sclerosis after long-term peritoneal dialysis treatment.
pubmed:affiliation
Department of Nephrology and Hypertension, Jikei University School of Medicine, Tokyo, Japan. mnakayama@jikei.ac.jp
pubmed:publicationType
Journal Article, Review