Source:http://linkedlifedata.com/resource/pubmed/id/16293795
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
2
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pubmed:dateCreated |
2006-1-20
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pubmed:abstractText |
The fibrous cap of the atherosclerotic lesion is believed to be critical to stability because disruption of the cap is the final event leading to plaque rupture. We have, therefore, used expression arrays to define the phenotype of the cap and other plaque components.
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pubmed:grant | |
pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Feb
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pubmed:issn |
1524-4636
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pubmed:author | |
pubmed:issnType |
Electronic
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pubmed:volume |
26
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
319-25
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading |
pubmed-meshheading:16293795-Carotid Arteries,
pubmed-meshheading:16293795-Carotid Artery Diseases,
pubmed-meshheading:16293795-Gene Expression Profiling,
pubmed-meshheading:16293795-Humans,
pubmed-meshheading:16293795-Immunohistochemistry,
pubmed-meshheading:16293795-Muscle, Smooth, Vascular,
pubmed-meshheading:16293795-Phenotype,
pubmed-meshheading:16293795-RGS Proteins,
pubmed-meshheading:16293795-Tunica Intima,
pubmed-meshheading:16293795-Tunica Media
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pubmed:year |
2006
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pubmed:articleTitle |
Expression profiling identifies smooth muscle cell diversity within human intima and plaque fibrous cap: loss of RGS5 distinguishes the cap.
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pubmed:affiliation |
Department of Pathology, Center for Cardiovascular Biology and Regenerative Medicine, University of Washington School of Medicine, Seattle, USA. ladams@u.washington.edu
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pubmed:publicationType |
Journal Article,
Research Support, N.I.H., Extramural
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