| pubmed-article:16263910 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:16263910 | lifeskim:mentions | umls-concept:C0162741 | lld:lifeskim |
| pubmed-article:16263910 | lifeskim:mentions | umls-concept:C0038435 | lld:lifeskim |
| pubmed-article:16263910 | lifeskim:mentions | umls-concept:C0037494 | lld:lifeskim |
| pubmed-article:16263910 | lifeskim:mentions | umls-concept:C0596988 | lld:lifeskim |
| pubmed-article:16263910 | lifeskim:mentions | umls-concept:C2346484 | lld:lifeskim |
| pubmed-article:16263910 | lifeskim:mentions | umls-concept:C1881189 | lld:lifeskim |
| pubmed-article:16263910 | pubmed:issue | 422 | lld:pubmed |
| pubmed-article:16263910 | pubmed:dateCreated | 2005-11-24 | lld:pubmed |
| pubmed-article:16263910 | pubmed:abstractText | The Arabidopsis thaliana ascorbate-deficient vtc-1 mutant has only 30% ascorbate contents of the wild type (WT). This ascorbate-deficient mutant was used here to study the physiological roles of ascorbate under salt stress in vivo. Salt stress resulted in a more significant decrease in CO2 assimilatory capacity in the vtc-1 mutant than in the WT. Photosystem II function in the Arabidopsis vtc-1 mutant also showed an increased sensitivity to salt stress. Oxidative stress, indicated by the hydrogen peroxide content, increased more dramatically in the vtc-1 mutant than in the WT under salt stress. To clarify the reason for the increased oxidative stress in the vtc-1 mutant, the contents of small antioxidant compounds and the activities of several antioxidant enzymes in the ascorbate-glutathione cycle were measured. Despite an elevated glutathione pool in the vtc-1 mutant, the ascorbate contents and the reduced form of ascorbate decreased very rapidly under salt stress. These results showed that the activities of MDAR and DHAR were lower in the vtc-1 mutant than in the WT under salt stress. Thus, low intrinsic ascorbate and an impaired ascorbate-glutathione cycle in the vtc-1 mutant under salt stress probably induced a dramatic decrease in the reduced form of ascorbate, which resulted in both enhanced ROS contents and decreased NPQ in the vtc-1 mutant. | lld:pubmed |
| pubmed-article:16263910 | pubmed:language | eng | lld:pubmed |
| pubmed-article:16263910 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16263910 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:16263910 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16263910 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16263910 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16263910 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16263910 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16263910 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16263910 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16263910 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16263910 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16263910 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16263910 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16263910 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16263910 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16263910 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16263910 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16263910 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16263910 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:16263910 | pubmed:issn | 0022-0957 | lld:pubmed |
| pubmed-article:16263910 | pubmed:author | pubmed-author:ZhangLixinL | lld:pubmed |
| pubmed-article:16263910 | pubmed:author | pubmed-author:GuoJinkuiJ | lld:pubmed |
| pubmed-article:16263910 | pubmed:author | pubmed-author:SuPeixiP | lld:pubmed |
| pubmed-article:16263910 | pubmed:author | pubmed-author:ChangXuexiang... | lld:pubmed |
| pubmed-article:16263910 | pubmed:author | pubmed-author:HuangChenghon... | lld:pubmed |
| pubmed-article:16263910 | pubmed:author | pubmed-author:HeWenliangW | lld:pubmed |
| pubmed-article:16263910 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:16263910 | pubmed:volume | 56 | lld:pubmed |
| pubmed-article:16263910 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16263910 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16263910 | pubmed:pagination | 3041-9 | lld:pubmed |
| pubmed-article:16263910 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:16263910 | pubmed:meshHeading | pubmed-meshheading:16263910... | lld:pubmed |
| pubmed-article:16263910 | pubmed:year | 2005 | lld:pubmed |
| pubmed-article:16263910 | pubmed:articleTitle | Increased sensitivity to salt stress in an ascorbate-deficient Arabidopsis mutant. | lld:pubmed |
| pubmed-article:16263910 | pubmed:affiliation | Cold and Arid Regions Environmental and Engineering Research Institute, Chinese Academy of Sciences, Lanzhou, 730000, China. | lld:pubmed |
| pubmed-article:16263910 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16263910 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| entrez-gene:818562 | entrezgene:pubmed | pubmed-article:16263910 | lld:entrezgene |
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