| pubmed-article:16165109 | rdf:type | pubmed:Citation | lld:pubmed |
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| pubmed-article:16165109 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
| pubmed-article:16165109 | lifeskim:mentions | umls-concept:C0026336 | lld:lifeskim |
| pubmed-article:16165109 | lifeskim:mentions | umls-concept:C1522564 | lld:lifeskim |
| pubmed-article:16165109 | lifeskim:mentions | umls-concept:C0085542 | lld:lifeskim |
| pubmed-article:16165109 | lifeskim:mentions | umls-concept:C0623362 | lld:lifeskim |
| pubmed-article:16165109 | lifeskim:mentions | umls-concept:C0038952 | lld:lifeskim |
| pubmed-article:16165109 | lifeskim:mentions | umls-concept:C0442805 | lld:lifeskim |
| pubmed-article:16165109 | lifeskim:mentions | umls-concept:C1150224 | lld:lifeskim |
| pubmed-article:16165109 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:16165109 | pubmed:dateCreated | 2006-1-30 | lld:pubmed |
| pubmed-article:16165109 | pubmed:abstractText | Oxidative stress is implicated in the pathogenesis of heart failure and affects the activity of matrix metalloproteinases (MMPs). We have now investigated the role of MMPs and their tissue inhibitors (TIMPs) in the transition from compensated left ventricular (LV) hypertrophy to heart failure as well as the effects of pravastatin on this transition in a rat model. | lld:pubmed |
| pubmed-article:16165109 | pubmed:language | eng | lld:pubmed |
| pubmed-article:16165109 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16165109 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:16165109 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:16165109 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16165109 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16165109 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:16165109 | pubmed:issn | 0008-6363 | lld:pubmed |
| pubmed-article:16165109 | pubmed:author | pubmed-author:KawanishiShos... | lld:pubmed |
| pubmed-article:16165109 | pubmed:author | pubmed-author:OikawaShinjiS | lld:pubmed |
| pubmed-article:16165109 | pubmed:author | pubmed-author:YamadaYoshiji... | lld:pubmed |
| pubmed-article:16165109 | pubmed:author | pubmed-author:ObataKojiK | lld:pubmed |
| pubmed-article:16165109 | pubmed:author | pubmed-author:NagataKohzoK | lld:pubmed |
| pubmed-article:16165109 | pubmed:author | pubmed-author:YokotaMitsuhi... | lld:pubmed |
| pubmed-article:16165109 | pubmed:author | pubmed-author:IchiharaGakuG | lld:pubmed |
| pubmed-article:16165109 | pubmed:author | pubmed-author:IchiharaSahok... | lld:pubmed |
| pubmed-article:16165109 | pubmed:author | pubmed-author:NodaAkikoA | lld:pubmed |
| pubmed-article:16165109 | pubmed:author | pubmed-author:XuJinglanJ | lld:pubmed |
| pubmed-article:16165109 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:16165109 | pubmed:day | 15 | lld:pubmed |
| pubmed-article:16165109 | pubmed:volume | 69 | lld:pubmed |
| pubmed-article:16165109 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16165109 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16165109 | pubmed:pagination | 726-35 | lld:pubmed |
| pubmed-article:16165109 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
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| pubmed-article:16165109 | pubmed:year | 2006 | lld:pubmed |
| pubmed-article:16165109 | pubmed:articleTitle | Pravastatin increases survival and suppresses an increase in myocardial matrix metalloproteinase activity in a rat model of heart failure. | lld:pubmed |
| pubmed-article:16165109 | pubmed:affiliation | Department of Cardiovascular Genome Science, Nagoya University School of Medicine, 65 Tsurumai-cho, Showa-ku, Nagoya 466-8550, Japan. | lld:pubmed |
| pubmed-article:16165109 | pubmed:publicationType | Journal Article | lld:pubmed |
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