Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
5
pubmed:dateCreated
1992-7-30
pubmed:abstractText
This study was performed to establish the relative efficiency of occlusive dressings and variable ambient temperature (group I) versus no dressings and variable ambient temperature (group II) versus no dressings and electromagnetic heaters (group III) for controlling the postburn hypermetabolic response. Fifteen burn patients and five normal controls (group IV) were studied when subjectively comfortable using partitional calorimetry, after which each patient was cold stressed by sequentially decreasing external energy support, and repeating calorimetry studies and serial plasma catecholamine assays. The percentage increase in heat production above predicted normal values was significantly increased for all groups when cold (C) versus neutral (N) (group I: [N] 24 +/- 24 versus [C] 49 +/- 25%; group II: [N] 46 +/- 35 versus [C] 74 +/- 47%; group III: [N] 21 +/- 20 versus [C] 78 +/- 25%; group IV: [N] -9 +/- 12 versus [C] 16 +/- 10%, p less than 0.05 all comparisons). Plasma catecholamine values did not increase significantly when patients were subjectively cold. These studies do not support the role of catecholamines as the primary mediator in the cause of the postburn hypermetabolic response. Using the patients' subjective comfort status as a guide for external energy support, it is possible to greatly reduce but not to eliminate the hypermetabolic response to burn injury.
pubmed:grant
pubmed:commentsCorrections
http://linkedlifedata.com/resource/pubmed/commentcorrection/1616385-1252123, http://linkedlifedata.com/resource/pubmed/commentcorrection/1616385-13017515, http://linkedlifedata.com/resource/pubmed/commentcorrection/1616385-1740794, http://linkedlifedata.com/resource/pubmed/commentcorrection/1616385-17758307, http://linkedlifedata.com/resource/pubmed/commentcorrection/1616385-2042096, http://linkedlifedata.com/resource/pubmed/commentcorrection/1616385-2108508, http://linkedlifedata.com/resource/pubmed/commentcorrection/1616385-2119089, http://linkedlifedata.com/resource/pubmed/commentcorrection/1616385-2257068, http://linkedlifedata.com/resource/pubmed/commentcorrection/1616385-2406054, http://linkedlifedata.com/resource/pubmed/commentcorrection/1616385-2494357, http://linkedlifedata.com/resource/pubmed/commentcorrection/1616385-2551907, http://linkedlifedata.com/resource/pubmed/commentcorrection/1616385-2889120, http://linkedlifedata.com/resource/pubmed/commentcorrection/1616385-3052328, http://linkedlifedata.com/resource/pubmed/commentcorrection/1616385-3672321, http://linkedlifedata.com/resource/pubmed/commentcorrection/1616385-4412350, http://linkedlifedata.com/resource/pubmed/commentcorrection/1616385-676332, http://linkedlifedata.com/resource/pubmed/commentcorrection/1616385-7235763, http://linkedlifedata.com/resource/pubmed/commentcorrection/1616385-759237
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
May
pubmed:issn
0003-4932
pubmed:author
pubmed:issnType
Print
pubmed:volume
215
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
485-90; discussion 490-1
pubmed:dateRevised
2010-9-9
pubmed:meshHeading
pubmed:year
1992
pubmed:articleTitle
Control of the hypermetabolic response to burn injury using environmental factors.
pubmed:affiliation
University of Arkansas Medical Sciences Campus, Department of Surgery, Little Rock 72205.
pubmed:publicationType
Journal Article, Clinical Trial, Comparative Study, Research Support, U.S. Gov't, P.H.S., Controlled Clinical Trial