Linked Life Data

16141465

Source:http://linkedlifedata.com/resource/pubmed/id/16141465

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
12
pubmed:dateCreated
2005-11-18
pubmed:abstractText
Reactive oxygen species are involved in many of the angiotensin II signalling pathways. We have thus investigated whether the angiotensin II type 1 (AT1) receptor antagonist, telmisartan, can inhibit the accelerated renal fibrosis and excess oxidative stress, which occurs after unilateral ureteral obstruction (UUO) in acatalasemic mice.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Dec
pubmed:issn
0931-0509
pubmed:author
pubmed:issnType
Print
pubmed:volume
20
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
2670-80
pubmed:dateRevised
2006-11-15
pubmed:meshHeading
pubmed-meshheading:16141465-Acatalasia, pubmed-meshheading:16141465-Angiotensin II Type 1 Receptor Blockers, pubmed-meshheading:16141465-Animals, pubmed-meshheading:16141465-Benzimidazoles, pubmed-meshheading:16141465-Benzoates, pubmed-meshheading:16141465-Disease Models, Animal, pubmed-meshheading:16141465-Fibrosis, pubmed-meshheading:16141465-Immunohistochemistry, pubmed-meshheading:16141465-In Situ Nick-End Labeling, pubmed-meshheading:16141465-Kidney, pubmed-meshheading:16141465-Kidney Diseases, pubmed-meshheading:16141465-Lipid Peroxidation, pubmed-meshheading:16141465-Male, pubmed-meshheading:16141465-Mice, pubmed-meshheading:16141465-Mice, Inbred C3H, pubmed-meshheading:16141465-Mice, Mutant Strains, pubmed-meshheading:16141465-NADPH Oxidase, pubmed-meshheading:16141465-Oxidative Stress, pubmed-meshheading:16141465-Treatment Outcome, pubmed-meshheading:16141465-Ureteral Obstruction
pubmed:year
2005
pubmed:articleTitle
Telmisartan inhibits both oxidative stress and renal fibrosis after unilateral ureteral obstruction in acatalasemic mice.
pubmed:affiliation
Department of Medicine and Clinical Science, Okayama University Graduate School of Medicine, Dentistry, and Pharmaceutical Sciences, 2-5-1 Shikata-cho, Okayama 700-8558, Japan. hitoshis@md.okayama-u.ac.jp
pubmed:publicationType
Journal Article, Comparative Study, Research Support, Non-U.S. Gov't