| pubmed-article:16128625 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:16128625 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
| pubmed-article:16128625 | lifeskim:mentions | umls-concept:C0033809 | lld:lifeskim |
| pubmed-article:16128625 | lifeskim:mentions | umls-concept:C0042765 | lld:lifeskim |
| pubmed-article:16128625 | lifeskim:mentions | umls-concept:C0282208 | lld:lifeskim |
| pubmed-article:16128625 | lifeskim:mentions | umls-concept:C2350024 | lld:lifeskim |
| pubmed-article:16128625 | lifeskim:mentions | umls-concept:C2349975 | lld:lifeskim |
| pubmed-article:16128625 | lifeskim:mentions | umls-concept:C0450254 | lld:lifeskim |
| pubmed-article:16128625 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:16128625 | pubmed:dateCreated | 2005-8-30 | lld:pubmed |
| pubmed-article:16128625 | pubmed:abstractText | We have shown previously that the PA-I lectin of Pseudomonas aeruginosa plays a key role in gut-derived sepsis during surgical stress. The aims of this study were to determine if the intestinal tract lumen of a stressed host contained soluble factors that could induce the expression of PA-I. | lld:pubmed |
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| pubmed-article:16128625 | pubmed:language | eng | lld:pubmed |
| pubmed-article:16128625 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16128625 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:16128625 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16128625 | pubmed:issn | 1096-2964 | lld:pubmed |
| pubmed-article:16128625 | pubmed:author | pubmed-author:HolbrookChris... | lld:pubmed |
| pubmed-article:16128625 | pubmed:author | pubmed-author:ChangEugene... | lld:pubmed |
| pubmed-article:16128625 | pubmed:author | pubmed-author:TurnerJerrold... | lld:pubmed |
| pubmed-article:16128625 | pubmed:author | pubmed-author:ZaborinaOlgaO | lld:pubmed |
| pubmed-article:16128625 | pubmed:author | pubmed-author:AlverdyJohn... | lld:pubmed |
| pubmed-article:16128625 | pubmed:author | pubmed-author:MuschMarkM | lld:pubmed |
| pubmed-article:16128625 | pubmed:author | pubmed-author:ZaborinAlexA | lld:pubmed |
| pubmed-article:16128625 | pubmed:author | pubmed-author:WuLicheng RLR | lld:pubmed |
| pubmed-article:16128625 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:16128625 | pubmed:volume | 6 | lld:pubmed |
| pubmed-article:16128625 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16128625 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16128625 | pubmed:pagination | 185-95 | lld:pubmed |
| pubmed-article:16128625 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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| pubmed-article:16128625 | pubmed:meshHeading | pubmed-meshheading:16128625... | lld:pubmed |
| pubmed-article:16128625 | pubmed:meshHeading | pubmed-meshheading:16128625... | lld:pubmed |
| pubmed-article:16128625 | pubmed:meshHeading | pubmed-meshheading:16128625... | lld:pubmed |
| pubmed-article:16128625 | pubmed:meshHeading | pubmed-meshheading:16128625... | lld:pubmed |
| pubmed-article:16128625 | pubmed:meshHeading | pubmed-meshheading:16128625... | lld:pubmed |
| pubmed-article:16128625 | pubmed:year | 2005 | lld:pubmed |
| pubmed-article:16128625 | pubmed:articleTitle | Surgical injury and metabolic stress enhance the virulence of the human opportunistic pathogen Pseudomonas aeruginosa. | lld:pubmed |
| pubmed-article:16128625 | pubmed:affiliation | Department of Surgery, University of Chicago, Chicago, Illinois 60637, USA. | lld:pubmed |
| pubmed-article:16128625 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16128625 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:16128625 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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