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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
2
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pubmed:dateCreated |
2005-8-30
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pubmed:abstractText |
We have shown previously that the PA-I lectin of Pseudomonas aeruginosa plays a key role in gut-derived sepsis during surgical stress. The aims of this study were to determine if the intestinal tract lumen of a stressed host contained soluble factors that could induce the expression of PA-I.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
|
|
pubmed:status |
MEDLINE
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pubmed:issn |
1096-2964
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pubmed:author |
|
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pubmed:issnType |
Print
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pubmed:volume |
6
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
185-95
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pubmed:dateRevised |
2008-11-21
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pubmed:meshHeading |
pubmed-meshheading:16128625-Adhesins, Bacterial,
pubmed-meshheading:16128625-Animals,
pubmed-meshheading:16128625-Cell Line,
pubmed-meshheading:16128625-Endotoxins,
pubmed-meshheading:16128625-Feces,
pubmed-meshheading:16128625-Humans,
pubmed-meshheading:16128625-Lectins,
pubmed-meshheading:16128625-Mice,
pubmed-meshheading:16128625-Models, Animal,
pubmed-meshheading:16128625-Pseudomonas Infections,
pubmed-meshheading:16128625-Pseudomonas aeruginosa,
pubmed-meshheading:16128625-Stress, Physiological,
pubmed-meshheading:16128625-Surgical Procedures, Operative,
pubmed-meshheading:16128625-Virulence
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pubmed:year |
2005
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pubmed:articleTitle |
Surgical injury and metabolic stress enhance the virulence of the human opportunistic pathogen Pseudomonas aeruginosa.
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pubmed:affiliation |
Department of Surgery, University of Chicago, Chicago, Illinois 60637, USA.
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.,
Research Support, N.I.H., Extramural
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