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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
10
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pubmed:dateCreated |
2005-10-3
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pubmed:abstractText |
Both advanced glycosylation end products (AGEs) and dendritic cells (DCs) have been shown to play a causative role in atherosclerosis. However, whether they function interactively in the process remains uncertain. We therefore studied the effects of AGE-bovine serum albumin (AGE-BSA) on the maturation of DCs and the expressions of scavenger receptor-A (SR-A) and receptor for AGEs (RAGE) on DCs.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
http://linkedlifedata.com/resource/pubmed/chemical/Anthracenes,
http://linkedlifedata.com/resource/pubmed/chemical/Antibodies,
http://linkedlifedata.com/resource/pubmed/chemical/Antigens, CD,
http://linkedlifedata.com/resource/pubmed/chemical/CD83 antigen,
http://linkedlifedata.com/resource/pubmed/chemical/Glycosylation End Products, Advanced,
http://linkedlifedata.com/resource/pubmed/chemical/Immunoglobulins,
http://linkedlifedata.com/resource/pubmed/chemical/Interleukin-12,
http://linkedlifedata.com/resource/pubmed/chemical/JNK Mitogen-Activated Protein...,
http://linkedlifedata.com/resource/pubmed/chemical/Membrane Glycoproteins,
http://linkedlifedata.com/resource/pubmed/chemical/Receptors, Cell Surface,
http://linkedlifedata.com/resource/pubmed/chemical/Receptors, Immunologic,
http://linkedlifedata.com/resource/pubmed/chemical/Scavenger Receptors, Class A,
http://linkedlifedata.com/resource/pubmed/chemical/Serum Albumin, Bovine,
http://linkedlifedata.com/resource/pubmed/chemical/advanced glycation end...,
http://linkedlifedata.com/resource/pubmed/chemical/advanced glycosylation end-product...,
http://linkedlifedata.com/resource/pubmed/chemical/anthra(1,9-cd)pyrazol-6(2H)-one
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pubmed:status |
MEDLINE
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pubmed:month |
Oct
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pubmed:issn |
1524-4636
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pubmed:author |
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pubmed:issnType |
Electronic
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pubmed:volume |
25
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
2157-63
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pubmed:dateRevised |
2009-11-19
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pubmed:meshHeading |
pubmed-meshheading:16100036-Anthracenes,
pubmed-meshheading:16100036-Antibodies,
pubmed-meshheading:16100036-Antigens, CD,
pubmed-meshheading:16100036-Atherosclerosis,
pubmed-meshheading:16100036-Cell Communication,
pubmed-meshheading:16100036-Cell Differentiation,
pubmed-meshheading:16100036-Dendritic Cells,
pubmed-meshheading:16100036-Gene Expression,
pubmed-meshheading:16100036-Glycosylation End Products, Advanced,
pubmed-meshheading:16100036-Humans,
pubmed-meshheading:16100036-Immunoglobulins,
pubmed-meshheading:16100036-Interleukin-12,
pubmed-meshheading:16100036-JNK Mitogen-Activated Protein Kinases,
pubmed-meshheading:16100036-MAP Kinase Signaling System,
pubmed-meshheading:16100036-Membrane Glycoproteins,
pubmed-meshheading:16100036-Monocytes,
pubmed-meshheading:16100036-Receptors, Cell Surface,
pubmed-meshheading:16100036-Receptors, Immunologic,
pubmed-meshheading:16100036-Scavenger Receptors, Class A,
pubmed-meshheading:16100036-Serum Albumin, Bovine,
pubmed-meshheading:16100036-T-Lymphocytes,
pubmed-meshheading:16100036-Up-Regulation
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pubmed:year |
2005
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pubmed:articleTitle |
Advanced glycosylation end products might promote atherosclerosis through inducing the immune maturation of dendritic cells.
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pubmed:affiliation |
Shanghai Institute of Cardiovascular Disease, Zhongshan Hospital, Fudan University, 180 Fenglin Rd, Shanghai 200032, People's Republic of China. gejunbo@zshospital.net
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pubmed:publicationType |
Journal Article,
In Vitro,
Research Support, Non-U.S. Gov't
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