Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
2
pubmed:dateCreated
2005-8-9
pubmed:abstractText
Moderate-sized studies have suggested that variants of candidate genes can influence laboratory markers of coronary artery disease (CAD), but whether they predict parallel changes in clinical CAD risk is unknown.
pubmed:grant
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
http://linkedlifedata.com/resource/pubmed/chemical/ATP binding cassette transporter 1, http://linkedlifedata.com/resource/pubmed/chemical/ATP-Binding Cassette Transporters, http://linkedlifedata.com/resource/pubmed/chemical/CETP protein, human, http://linkedlifedata.com/resource/pubmed/chemical/Carrier Proteins, http://linkedlifedata.com/resource/pubmed/chemical/Cholesterol, HDL, http://linkedlifedata.com/resource/pubmed/chemical/Cholesterol Ester Transfer Proteins, http://linkedlifedata.com/resource/pubmed/chemical/Glycoproteins, http://linkedlifedata.com/resource/pubmed/chemical/Lipase, http://linkedlifedata.com/resource/pubmed/chemical/Lipoprotein Lipase, http://linkedlifedata.com/resource/pubmed/chemical/Plasminogen Activator Inhibitor 1, http://linkedlifedata.com/resource/pubmed/chemical/Triglycerides, http://linkedlifedata.com/resource/pubmed/chemical/hepatic lipase, human
pubmed:status
MEDLINE
pubmed:month
Aug
pubmed:issn
1097-6744
pubmed:author
pubmed:issnType
Electronic
pubmed:volume
150
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
243-50
pubmed:dateRevised
2007-11-14
pubmed:meshHeading
pubmed-meshheading:16086925-ATP-Binding Cassette Transporters, pubmed-meshheading:16086925-Aged, pubmed-meshheading:16086925-Alleles, pubmed-meshheading:16086925-Carrier Proteins, pubmed-meshheading:16086925-Case-Control Studies, pubmed-meshheading:16086925-Cholesterol, HDL, pubmed-meshheading:16086925-Cholesterol Ester Transfer Proteins, pubmed-meshheading:16086925-Comorbidity, pubmed-meshheading:16086925-Coronary Angiography, pubmed-meshheading:16086925-Coronary Disease, pubmed-meshheading:16086925-Epistasis, Genetic, pubmed-meshheading:16086925-Female, pubmed-meshheading:16086925-Gene Frequency, pubmed-meshheading:16086925-Genetic Predisposition to Disease, pubmed-meshheading:16086925-Genotype, pubmed-meshheading:16086925-Glycoproteins, pubmed-meshheading:16086925-Humans, pubmed-meshheading:16086925-Lipase, pubmed-meshheading:16086925-Lipoprotein Lipase, pubmed-meshheading:16086925-Male, pubmed-meshheading:16086925-Middle Aged, pubmed-meshheading:16086925-Phenotype, pubmed-meshheading:16086925-Plasminogen Activator Inhibitor 1, pubmed-meshheading:16086925-Polymerase Chain Reaction, pubmed-meshheading:16086925-Polymorphism, Restriction Fragment Length, pubmed-meshheading:16086925-Polymorphism, Single Nucleotide, pubmed-meshheading:16086925-Predictive Value of Tests, pubmed-meshheading:16086925-Prospective Studies, pubmed-meshheading:16086925-Risk Factors, pubmed-meshheading:16086925-Triglycerides
pubmed:year
2005
pubmed:articleTitle
Candidate gene susceptibility variants predict intermediate end points but not angiographic coronary artery disease.
pubmed:affiliation
Cardiovascular Department, LDS Hospital, Salt Lake City, UT 84143, USA.
pubmed:publicationType
Journal Article, Research Support, U.S. Gov't, P.H.S., Research Support, Non-U.S. Gov't, Evaluation Studies, Research Support, N.I.H., Extramural